hypersensitivity

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hy·per·sen·si·tive

 (hī′pər-sĕn′sĭ-tĭv)
adj.
1. Highly or excessively sensitive.
2. Responding excessively to the stimulus of a foreign agent, such as an allergen.

hy′per·sen′si·tive·ness, hy′per·sen′si·tiv′i·ty n.
hy′per·sen′si·tize′ (-tīz′) v.

hypersensitivity

extreme or abnormal sensitivity, as to criticism. — hypersensitive, adj.
See also: Psychology
extreme or abnormal sensitivity, as to criticism. — hypersensitive, adj.
See also: Attitudes
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.hypersensitivity - pathological sensitivity
predisposition, sensitivity - susceptibility to a pathogen
cryaesthesia, cryesthesia - hypersensitivity to cold
hypersensitivity reaction - an inappropriate and excessive reaction to an allergen (as pollen or dust or animal hair or certain foods); severity ranges from mild allergy to severe systemic reactions leading to anaphylactic shock
2.hypersensitivity - extreme sensitivity
sensitivity, sensitiveness, sensibility - (physiology) responsiveness to external stimuli; the faculty of sensation; "sensitivity to pain"
Translations

hypersensitivity

n hipersensibilidad f
References in periodicals archive ?
Induction of Tcell mediated delayed type hypersensitivity, and rapid production of T cells in mice was reported previously (Titus 1991).
The effects of phyllanthin on cellular immune responses in treated /non treated mice were determined by measuring CD 11b/CD 18 integrin expression, phagocytosis, nitric oxide (NO) production, myeloperoxidase activity (MPO), T and B cells proliferation, lymphocyte phenotyping, serum cytokines production by activated T-cells and delayed type hypersensitivity (DTH).
The higher cellular immune responses in administered birds might be due to the delayed type hypersensitivity and/or stimulatory effects of biomolecules on macrophages that may lead to increase in thickness of toe web in response to PHA-P and DNCB (T-cell mitogens).
Delayed type hypersensitivity to fungi was identified and described long ago.
Present study describes the effects of ketoprofen on cell mediated immunity as measured through macrophage engulfment, nitric oxide (NO) detection, cyclophosphamide induced neutropenia and delayed type hypersensitivity assays following injection of 1mg/kg and 5mg/kg of the drug into the mice.
Additionally, O3 decrease the production of other inflammatory mediators (such as TNF[alpha]), reduce T cell receptor expression, immunoglobulin A (IgA) expression, antigenic stimulation responses by lymphocytes, delayed type hypersensitivity response and accessory cell function.
The immunity generated by AE37 appeared to be long lasting, as significant vaccine-specific delayed type hypersensitivity (DTH) reactions were still apparent 6 months after the end of vaccinations.
The Th-1 cells secrete interferon-gamma (IFN-[alpha]), interleukin-2 (IL-2) and tumour necrosis factor-beta (TNF-[beta]) and are responsible for cell-mediated inflammatory reactions, delayed type hypersensitivity and tissue injury in infections and autoimmune diseases.