A computed tomography scan of his head identified several infarcts in the territories of the left middle cerebral artery, the left and right posterior cerebral
artery, and lacunar infarcts in the left basal ganglia and right thalamus.
The right vertebral artery, basilar artery, bilateral anterior cerebral arteries, middle cerebral arteries, and posterior cerebral
arteries were not narrowed.
Hemiplegia Vegetativa Alterna (HVA) is the clinical condition characterized with ipsilateral Horner's syndrome (HS) and contralateral hemi-hyperhidrosis and is mainly used to define the condition at stroke patients with posterior cerebral
artery (PCA) or middle cerebral artery (MCA) occlusion (1,2).
Dominant arterial supply is via the posterior cerebral
arteries and distal branches of the basilar artery (Figures 4, 6, and 7).
The modest prevalence of involvement of the ahPCG reflects two factors: (1) even with MCA territory infarcts, the bulk of the lesion is often subcortical, reflecting either occlusion of multiple lenticulostriate vessels by clot in the M1 portion, or infarction of the lenticulostriate endzone (a "desert zone") when clot wedges at the carotid T-junction , and (2) even when the cortex is involved, the lesion is characteristically perisylvian and only sometimes extends sufficiently dorsally to involve the ahPCG, reflecting the degree to which posterior cerebral
artery-MCA and ACA-MCA end to end anastomoses serve to save cortex from infarction.
Vascular syndromes involving the posterior cerebral
artery characteristically involve a contralateral visual field deficit.
then passes between the posterior cerebral
and superior cerebellar arteries
Pure superficial posterior cerebral
artery territory infarction in the Lausanne Stroke Registry.
RPLS symptoms include headache, seizures, confusion, and visual changes with characteristic neuroimaging findings of posterior cerebral
white matter edema.
In addition, multiple focal areas of narrowing affecting bilateral anterior, middle and posterior cerebral
arteries indicated possible vasculitis.
9%) followed by posterior cerebral
artery 13%, brainstem 13%, anterior cerebral artery lesion in 11.
A follow-up MRI scan showed new compression of the non-treated right cerebral penduncle by the opposite posterior cerebral