If the patient has adequate defecation and minimal gastrointestinal decompression, the intestinal tract is not affected by intestinal atresia, intestinal malrotation, or other complications.
found elevated DEFA5 and DEFA6 mRNA levels in infants with NEC compared with five near-term controls (four patients with intestinal atresia and one with meconium ileus) [20].
[1] Intestinal atresia is a common congenital gut disease [2] detected by prenatal ultrasonography in second to third trimester and confirmed at birth by intestinal obstruction.
We would like to share our experience with a similar case, in a child with a prenatal diagnosis of partial agenesis of the corpus callosum and multiple intestinal atresia, born at 37 weeks' gestational age, who developed postnatal hypoxic-ischaemic encephalopathy (HIE).
([section][section][section]) Esophageal atresia (750.3), rectal and large intestinal atresia/stenosis (751.2), pyloric stenosis (750.5 among all ages; 537.0 among infants aged <1 year), Hirschsprung disease (751.3), biliary atresia (751.61), or small intestinal atresia (751.1).
The differential diagnosis of antenatal intra-abdominal cysts includes ovarian cysts, renal cysts, choledochal cysts, hepatic cysts, mesenteric or omental cysts, and dilated bowel loops of intestinal atresia [3].
Other common causes of SBS in the United States include resection following intestinal atresia, dysmotility disorder, gastroschisis, or other congenital malformations including midgut volvulus from malrotation [8, 9].
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