It is used to understand the dynamic processes and cellular structures, to study endocytosis and
exocytosis, cellular integrity, enzyme activity, protein trafficking and localization of molecules.
Synapsins and syntaxins are vesicle proteins that exhibit principal roles in
exocytosis and in presynaptic terminals in the clinical symptoms of experimental autoimmune encephalomyelitis (11).
The surface of the tumor showed focally intense neutrophilic
exocytosis of the epithelium with necrosis associated with granulation and inflammatory tissue; necrosis was not seen elsewhere within the neoplasm.
BTX-A inhibits the
exocytosis of acetylcholine on cholinergic nerve endings of motor nerves and blocks the release of acetylcholine by the neuron.
vivax malaria also has pathogenic mechanisms, such as increased inflammatory response and cytokine production, Weibel-Palade bodies
exocytosis, altered thrombosis, platelet activation, and impairment of vasomotor responses as seen with P.
In the dermis, there is a perivascular infiltrate with
exocytosis of neutrophils and lymphocytes, along with vasodilatation and edema.
Ten genes (ATP8A1, bridging integrator 2, CD93 molecule [CD93], exophilin 5, GRB2 associated binding protein 2, n-ethylmaleimide-sensitive factor attachment protein, beta, PLA2G6, PTPRC, and vesicle associated membrane protein 1 [VAMP1]) were associated with
exocytosis, and five genes (ATP8A1, CD93, DMBT1, PTPRC, and VAMP1) were classified under the secretory granule membrane term in the GO network.
Calcium is an important intracellular messenger, triggers many cellular signaling pathways in neurons associated with differentiation and growth,
exocytosis, synaptic transmission and synaptic plasticity during learning and memory (2).
This protein, existing in various organs, is involved in intercellular synaptic transmission, vesicular
exocytosis and general secretion.
Classically, epidermotropism (single-cell
exocytosis of lymphocytes into the epidermis) is present.
Exocytosis could be observed in pityriasis rosea, lichen planus, and seborrheic dermatitis.
When levels of Ca+ are sufficiently elevated during rapid neuronal firing, the calcium signaling activates the transcription of IGF binding proteins [63] as well as releases IGF-1 is released via
exocytosis, further modifying IGF-1 signaling [64].