Acquired left ventricular pseudoaneurysms develop after transmural myocardial infarction (55% in reviews), surgery (33%), trauma (7%), or infection (5%).[4] Rupture of the left ventricle after myocardial infarction usually leads to acute pericardial tamponade and immediate death.
Life-threatening complications, including outflow tract obstruction, coronary insufficiency, heart failure, pericardial tamponade, thromboembolia, dysrhythmia, and even sudden cardiac death, can be noticed (1-5).
The symptoms for the Acute aortic dissection range from pain related to the acute event and collapse due to aortic rupture or pericardial tamponade (this happens when fluid in the pericardium -- the sac around the heart -- builds up and results in compression of the heart).
We suggest that clinicians remain alert for unusual complications of influenza infection for patients presenting with shock physiology, including pericardial tamponade and myocarditis.
Severe complications occurred in two of the 11 patients receiving transvenous management, with one patient dying 10 days after lead extraction [6] and the other developing pericardial tamponade after transvenous lead extraction that required pericardiocentesis [7].
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