adenomatous


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ad·e·no·ma

 (ăd′n-ō′mə)
n. pl. ad·e·no·mas or ad·e·no·ma·ta (-mə-tə)
A benign epithelial tumor having a glandular origin and structure.

ad′e·nom′a·toid′ (ăd′n-ŏm′ə-toid′) adj.
ad′e·nom′a·tous (-ŏm′ə-təs) adj.

adenomatous

(ˌædɪˈnəʊmətəs)
adj
characterized by a collection of glandular growths
Translations

adenomatous

adj adenomatoso
References in periodicals archive ?
Mesenteric and abdominal wall desmoid tumors are usually associated with familial adenomatous polyposis, which is caused by germline mutations in the adenomatous polyposis coli gene.
Non-neoplastic and non-inflammatory lesion: Colloid goiter, adenomatous goiter, benign cystic lesion, hyperplasia, thyroglossal cyst, thyrotoxicosis.
Basil Morson from London, United Kingdom, perhaps the best known and revered gastrointestinal pathologist of the mid to late twentieth century, used the term metaplastic polyp in 1962 for what we now call hyperplastic polyps in order to distinguish them from true adenomatous proliferations.
Histopathology of the polyps showed adenomatous changes with stromal edema and dilated hyperplastic glands (Figures 8 and 9).
Adenomatous goiter was the commonest non neoplastic lesion, follicular adenoma was the commonest benign neoplastic lesion where is papillary carcinoma was the commonest malignant lesion.
USPRwire, Wed Sep 23 2015] GlobalData's clinical trial report, "Familial Adenomatous Polyposis Coli Global Clinical Trials Review, H2, 2015" provides an overview of Familial Adenomatous Polyposis Coli clinical trials scenario.
MICROSCOPY: Section revealed a polyp with central fibrovascular core where the lining exhibited both villous and adenomatous components.
average risk of developing colorectal cancer (no personal history of adenomatous polyps, of colorectal cancer, or inflammatory bowel disease, including Crohn's Disease and ulcerative colitis; no family history of colorectal cancers or an adenomatous polyp, familial adenomatous polyposis, or hereditary nonpolyposis colorectal cancer).
In addition to an association with low birth weight, there are several linked genetic diseases including overgrowth syndromes such as Beckwith-Wiedemann syndrome, chromosomally linked conditions (trisomies 2, 8 and 20) and X-linked Simpson-Golabi-Behmel syndrome, type 1 glycogen storage diseases, Li-Fraumeni syndrome, familial adenomatous polyposis (FAP) (3-6) and type 1 neurofibromatosis.
pylori to rise with the increasing number, as well as the size, of adenomatous polyps.
The company also announced dosing of the first patient in Cohort 2 in the Dose Escalation Phase of the START-FAP (Safety and Tolerability of An RNAi Therapeutic in Familial Adenomatous Polyposis) clinical trial with CEQ508.