expression with molecular markers discriminates invasiveness in growth hormone and prolactin pituitary adenomas.
The topics include the integration of cadherin
adhesion and cytoskeleton at adherens junction, making connections: guidance cues and receptors at non-neural cell-cell-junctions, tuning collective cell migration by cell-cell junction regulation, beyond cell-cell adhesion: sensational cadherins
for hearing and balance, and the loss of E-cadherin
-dependent cell-cell adhesion and the development and progression of cancer.
Numerous isoforms of cadherin
have been identified in a tissue-specific manner and shown to regulate morphogenesis through homophilic binding (Nose et al.
Immunoregulatory functions of KLRG1 cadherin
interactions are dependent on forward and reverse signaling.
The loss of E- Cadherin
, alpha- and beta- catenin expression is associated with metastasis and poor prognosis in invasive breast cancer.
In first, to induce toxicity the sequential binding takes place with multiple receptors, while according to the second, binding to the cadherin
stimulates an intracellular reactions cascade leading to the toxicity in cell.
The GLE suppressed the TGF-[beta]1 -induced morphological changes and the changes in cadherin
expression, and also inhibited the formation of F-actin stress fibers, which are a hallmark of EMT.
is a member of the cadherin
superfamily, serving as the main epithelial intercellular adhesion molecule.
switching in human prostate cancer progression.
EPLIN mediates linkage of the cadherin
catenin complex to F-actin and stabilizes the circumferential actin belt.
Among them is the Human Epithelial Cadherin
), a calcium-dependent trans membrane glycoprotein directly involved in the process of cell adhesion.
, a specific marker for the distal portion of the nephron and related renal neoplasms.