caudate nucleus

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caudate nucleus

n.
A basal ganglion located in the lateral ventricle of the brain that has a curved, taillike extension and functions in motor control.
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Noun1.caudate nucleus - a tail-shaped basal ganglion located in a lateral ventricle of the brain
basal ganglion - any of several masses of subcortical grey matter at the base of each cerebral hemisphere that seem to be involved in the regulation of voluntary movement
corpus striatum, striate body, striatum - a striped mass of white and grey matter located in front of the thalamus in each cerebral hemisphere; consists of the caudate nucleus and the lenticular nucleus
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References in periodicals archive ?
One case had bilateral basal ganglia involvement and the other had bilateral symmetric T2WI and FLAIR hyperintensities in bilateral caudate nuclei, lentiform nuclei, ventrolateral thalami, mid brain, periaqueductal gray matter and the pons.
Release of dopamine evoked by electrical stimulation of the motor and visual areas of the cerebral cortex in both caudate nuclei and in the substantia nigra in the cat.
Brain computed tomography (CT) showed symmetrical hypodensities in the caudate nuclei and magnetic resonance imaging (MRI) showed an increased signal on T2-weighted/fluid attenuation inversion recovery (FLAIR) sequences in the same region, although extending to involve the anterior lentiform nuclei (Fig.
Differential expression of DSCR in human brain A quantitative analysis of global transcription of DSCR genes along the structures of the brain nuclei and the limbic lobe revealed that the highest values of z-score were registered in the area that includes the putamen for DSCR3, RCAN1 as well as in left and right caudate nuclei for DSCR6, and the left and right globus pallidus for DSCR3, SH3GBR, DYRK1A, CLIC6 and PRMT2.
CT features in early infantile Krabbe disease are increased attenuation in cerebellum, brainstem, thalami, caudate nuclei and corona radiata on non-contrast CT.
Brain MRI revealed bilateral FLAIR and T1 hyperintensities of caudate nuclei, putamina and frontal lobe involving most of the anterior cingulate gyrus and superior frontal gyrus bilaterally, with restricted diffusion (Figure 1).
In neurologically symptomatic patients, T2 hyperintensity is noted in the putamina, caudate nuclei, thalami, midbrain and pons.