daminozide


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da·min·o·zide

 (də-mĭn′ə-zīd′)
n.
A chemical plant growth regulator, C6H12N2O3, formerly used to increase the storage life of fruit, especially apples, and currently used to control growth and flowering of commercially grown ornamental plants.

[d(imethyl) + amino- + (hydra)z(ine) + -ide.]

daminozide

(dəˈmɪnəˌzaɪd)
n
(Horticulture) another name for Alar
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.daminozide - a chemical sprayed on fruit trees to regulate their growth so the entire crop can be harvested at one timedaminozide - a chemical sprayed on fruit trees to regulate their growth so the entire crop can be harvested at one time
chemical, chemical substance - material produced by or used in a reaction involving changes in atoms or molecules
References in periodicals archive ?
Effects of BA, GA 4+7, and daminozide on fruit set, fruit quality, vegetative growth, flower initian, and flower quality of 'Golden Delicius' apple.
The "Alar Scare" in 1989 involved the use of the chemical daminozide (Alar) which keeps fruit firm and full-colored beyond its natural shelf-life (Egan, 1991).
Back in 1989, another apple-related chemical, daminozide, dominated the headlines.
In 1989 actress Meryl Streep appeared on "60 Minutes" and said the use of the growth regulator Daminozide, commonly known es Alar, on apples grown for human consumption would cause cancer.
Drought, paclobutrazol, abscisic acid, and gibberellic acid as alternatives to daminozide in tomato transplant production.
Pisarczyk JM, Splittstoesser WE (1979) Controlling Tomato Transplants Height with Chlormequat, Daminozide and Etephon.
Interaction between foliar potassium and applied daminozide, chlormequat chloride, and chlorflurecolmethyl.
Domestically, 1990 marked the conclusion of a regulatory battle over the pesticide daminozide (known by its trade name, Alar) in which the Environmental Protection Agency (EPA), the Uniroyal Corporation (Alar's chief manufacturer), and environmental groups each contested the safety of Alar through opposing risk-based conclusions involving competing exposure models and dose-response relationships.