4) Glycation also can affect elastin: Recent research suggests that glycation can engender elastosis
, which is elastin that is abnormally clumped together and presents more frequently in aged skin.
Enrolled study subjects will receive one procedure with the Company's J-Plasma technology at enrollment and wrinkle severity will be assessed using the Fitzpatrick Wrinkle and Elastosis
Scale at baseline and at each follow-up time point.
5-7,16) In a six-week period after injection, chronic cellular infiltration is accompanied by focal diffuse elastosis
, thickening and disorganized collagen fibers, and elastin fragmentation.
Dermatoheliosis is a combination of skin changes produced by solar damage and consists of senile comedones, irregular pigmentation, wrinkling, scaling actinic keratosis, elastosis
To the Editor: Elastosis
perforans serpiginosa (EPS) is part of different kinds of perforating diseases that are dependent on factors such as idiopathic, reactive, or drug induced.
Sin embargo, estudios histopatologicos han descartado dicha presencia, al encontrar solo elastosis
de los tejidos (21).
Exposure to sun not only causes pre-cancerous (actinic keratosis) and cancerous (basal cell carcinoma, squamous cell carcinoma and melanoma) skin lesions - caused by loss of the skin's immune function, it also leads to benign tumors, fine and coarse wrinkles, freckles, sallowness - a yellow discoloration of the skin, telangiectasias - the dilation of small blood vessels under the skin, and elastosis
- the destruction of the elastic tissue causing lines and wrinkles.
Lax skin, pigmentation, elastosis
and wrinkles are signs of photo-damage.
Rest of cutaneous examination revealed signs of solar damage like multiple seborrheic keratoses and solar elastosis
Ultraviolet radiation can damage the skin in a number of ways such as sunburn, actinic keratosis, actinic elastosis
, actinic cheilitis, triradiaite scars, senile comedones, purpura, poikiloderma and skin cancer.
A definite diagnosis of PPFE is made based on the constellation features of upper zone fibrosis of the visceral pleura (described as intense by Kusagaya et al (10); Figures 1 and 2); prominent homogenous subpleural intra-alveolar fibrosis with alveolar septal elastosis
(Figure 3); sparing of the parenchyma away from the pleura (Figure 1); scant, patchy lymphoplasmacytic infiltrates; and small numbers, at the very most, of fibroblastic foci.
Specifically, histologically, there was no granulomatous disease or lymphocytic interstitial pneumonia pattern present suggestive of granulomatous-lymphocytic interstitial lung disease (GLILD) and there was no interstitial elastosis
suggestive of pleuroparenchymal fibroelastosis (PPFE).