References in periodicals archive ?
Fusion of the transmembrane protease serine 2 gene and ETS-related gene (TMPRSS2-ERG) is the most common molecular subtype of erythroblast transformation-specific (ETS) family gene fusions, occurring in approximately 50% of prostate carcinomas and about 20% of high-grade prostatic intraepithelial neoplasias.
Examination of maternal plasma erythropoietin and activin A concentrations with regard to circulatory erythroblast levels in normal and preeclamptic pregnancies.
However, it is important that marginal results in the induction of MN be taken with reservation, since there is evidence that compound-related disturbances in rodent physiology, such as body temperature and erythroblast toxicity, can also modify MN frequencies and increase erythropoiesis by stimulating cell division in bone marrow and peripheral blood [39].
myeloblast, monoblast, erythroblast and megakaryoblast that accumulate in the marrow and interfere with production of blood cells.
Anaemia may be exacerbated by bone marrow suppression, with evidence of malaria-associated dyserythropoiesis, premature erythroblast and stem cell death, and impaired haem synthesis.
To eliminate this possibility, the researchers zeroed in on a red blood cell, called an erythroblast, that has a brief life span.
The tumor was diffusely immunoreactive for vimentin and focally positive for CD31 and erythroblast transformationspecific transcription factor (ERG).
The diagnosis of CDA can be made with high specificity from morphological analysis alone by light microscopy as the findings such as erythroblast nuclei abnormal.
Though anemia during Trypanosoma cruzi is not accompanied by a decrease in reticulocyte numbers, similar to our findings, chemical inhibition of nitric oxide or treatment with anti-TNF did not rescue mice from anemia even though these treatments resulted in increased numbers of reticulocytes and suppressed decreases in bone marrow erythroblast numbers during infection.
The sideroblastic anaemias are a heterogenous group of disorders with the unique characteristic of amorphous iron deposits in erythroblast mitochondria (1).
Cytokines such as IL-1, IL-6, TNF-[alpha], INF-[gamma], and lipopolysaccharide stimulate a decrease in the iron supply to RBC precursors, primarily at the erythroblast stage of development (Weiss & Goodnough, 2005).