hypercatabolism


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hypercatabolism

(ˌhaɪpəkəˈtæbəˌlɪzəm)
n
(Medicine) med an abnormally high metabolic breakdown of a substance or tissue which leads to weight loss and physical deterioration
References in periodicals archive ?
Cytokines, atherogenesis, and hypercatabolism in chronic kidney disease: a dreadful triad.
These findings may emphasize the fact that calpains are activated earlier during cachexia related to gastric cancer, before substantial weight loss and hypercatabolism of skeletal muscle by the UPS.
The cause of malnutrition in dialysis patients is not very clear, but some probable causes are inadequate nutrient intake, anorexia, dietary restrictions, nutrient losses during dialysis, hypercatabolism caused by comorbid illness or associated with dialysis treatment, endocrine disorders of uremia etc.
In ICU patients, energy and protein stores decrease because of hypercatabolism.
It has been suggested that hypermetabolism after a neurotrauma may be initiated by injured or dead tissue, perfusion deficits, or infection (Donaldson, Borzatta, & Matossian, 2000) and that hypercatabolism is related to the severity of the injury and secondary release of hormones such as insulin, glucagon, cortisol, epinephrine, and norepinephrine (Vizzini & Aranda-Michel, 2011).
Excessive elaboration of proinflammatory cytokines are involved in cachexia-related hypercatabolism.
In addition, increased BUN concentrations may be seen with increased dietary protein intake, hypercatabolism, corticosteroid use, or gastrointestinal bleeding.
Insufficient nutrient intake, nutrient losses during dialysis, co-morbid illness-induced hypercatabolism (such as cardiovascular disease, infection), and/or dialysis treatments are among the implicated underlying factors.
In the present study, during the period of 1985 to 1996 the type of dialysis indicated was intermittent peritoneal dialysis (IPD) but in case of extreme hypercatabolism where the removal of urea was inadequately performed by IPD classic hemodialysis or continuous slow hemodialysis in hemodynamically unstable patients were indicated.
Immune complexes and complement hypercatabolism in patients with leprosy.