natriuresis


Also found in: Thesaurus, Medical, Encyclopedia, Wikipedia.

na·tri·u·re·sis

 (nā′trə-yo͝o-rē′sĭs)
n.
Excretion of excessive amounts of sodium in the urine.

[New Latin natriūrēsis : natrium, sodium (from French natron, natron; see natron) + ūrēsis, urination (from Greek ourēsis, from ourein, to urinate; see uretic).]

na′tri·u·ret′ic (-rĕt′ĭk) adj.

natriuresis

(ˌneɪtrɪjʊˈriːsɪs)
n
(Medicine) the presence of a large amount of sodium in urine

na•tri•u•re•sis

(ˌneɪ trə yʊˈri sɪs)

n.
excretion of sodium in the urine.
[1957; natrium sodium (< German; see natron) + -uresis < Greek oúrēsis urination =ourē-, variant s. of oureîn to urinate + -sis -sis]
na`tri•u•ret′ic (-ˈrɛt ɪk) adj., n.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.natriuresis - the presence of abnormally large amounts of sodium in the urine
symptom - (medicine) any sensation or change in bodily function that is experienced by a patient and is associated with a particular disease
Mentioned in ?
References in periodicals archive ?
BNP is a neurohormone that acts to relieve the symptoms associated with volume expansion and pressure overload by promoting natriuresis and diuresis, vasodilation, and the suppression of the renin angiotensin aldosterone system.
The drinking water (base solution) contained 3 g/L NaCl in distilled water to overcome natriuresis in diabetic rats.
Both atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP) induce vasodilatation, natriuresis and diuresis.
This can lead to a natriuresis, the excessive urinary excretion of sodium causing symptomatic hyponatremia and orthostatic hypotension (Hamdi et al.
SGLT2 inhibitors also cause natriuresis (urinary excretion of sodium) and weight loss, and are associated with an antihypertensive effect through sodium cotransport (Wanner, 2017).
The effects are of short duration and system reset to the blood pressure levels necessary to maintain pressure in kidney necessary for natriuresis.
So if nighttime blood pressure is high it could lead through increased pressure natriuresis to increased urine production.
By causing the release of prostaglandins and nitric oxide (NO), the passage of BK into the renal arteries increases the blood flow to the kidneys and causes diuresis and natriuresis (23, 24).
Atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP) were described in the 1980s as circulating peptides released from the heart and involved in maintaining cardiorenal homeostasis by diuresis and natriuresis (3).
2] and muscarinic receptors blockade prevents diuresis, natriuresis, kaliuresis, and chloride excretion induced by ES-EC
The result of this study could be explained by understanding the physiological actions of BNP which include decrease in vascular resistance as well as an increase in natriuresis.