steatosis


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ste·a·to·sis

 (stē′ə-tō′sĭs)

steatosis

(ˌstɪəˈtəʊsɪs)
n
the abnormal accumulation of fat within a cell or organ
Translations
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9) In a meta-analysis of 3064 patients in 13 studies, NAFLD Fibrosis Score could effectively predict and exclude advanced fibrosis and may be helpful along with other blood work (FibroSure or FIBROSpect) in determining the need for liver biopsy when ultrasound is not definitive (17) Elevated ferritin, as in the general population, is also a risk factor for steatosis in those with HCV infection.
The purpose of the present study was to evaluate whether suboptimal anti-HCV treatment that did not achieve sustained virological response (SVR) influenced the progression of liver fibrosis, steatosis and necroinflammation between 1st and 2nd LB.
They consider such aspects as what non-alcoholic fatty liver disease (NAFLD) is and why it is important, why the connection between NAFLD and diabetes is important, the clinical importance and therapeutic implications of physical activity and cardiovascular fitness in patients with NAFLD, NAFLD in South America and Hispanic people, whether alcohol in NAFLD is an oxymoron or a new standard of care, and metabolic factors and steatosis in patients with hepatitis B and C.
In addition, reduced adiponectin levels commonly associated with obesity may establish a proinflammatory factor, thus increasing vulnerability to lipotoxicity, which promotes progression from simple steatosis (i.
SAN FRANCISCO--Postmenopausal status is independently associated with a twofold increased risk of hepatic steatosis, the Dallas Heart Study has shown.
They were subjected to liver biopsies which were assessed for grade, stage, steatosis, stainable iron and characteristic histological lesions.
Grade 3-severe steatosis with higher reduction in beam penetration and loss of echos even from the main branches of the portal veins, and large echo difference between the kidney and liver (11), (12).
OA induced hepatic steatosis in HepG2 cells was marked by significant accumulation of lipid droplets as determined by Oil-Red-0 (ORO) based colorimetric assay, increased triacylglycerol (TAG) and increased lipid peroxidation.
The 306 participants were asked about caffeine coffee consumption and categorized into four groups: patients with no sign of fibrosis on ultrasound (control), steatosis, NASH stage 0-1, and NASH stage 2-4.
The spectrum of NAFLD ranges from simple steatosis to the development of necro-inflammmatory change (non-alcoholic steatohepatitis [NASH]) to the formation of fibrosis and cirrhosis.
Among the total study subjects, the proportion of subjects with mild steatosis was 19.
Steatosis may occur in 90 percent of individuals who consume more than 60 g/d of alcohol (or about five drinks per day) (Crabb 1999).