NSAID classification Non-selective inhibition Acetic acid Proprionic acid Fenamate derivatives derivatives derivatives * Diclofenac * Fenoprofen * Meclofenamate * Etodolac * Flurbiprofen * Mefenamic acid * Indomethacin * Ibuprofen * Sulindac
* Ketoprofen * Tolmetin * Naproxen * Oxaprozin Naphthyl alkanone Salicylates * Nabumetone * Aspirin * Diflunisal * Choline magnesium trisalicylate * Salicylate Enolic acid derivatives * Piroxicam * Meloxicam Selective COX-2 inhibition * Celecoxib * Rofecoxib Table 9.
Their findings were released today in an article titled "Pharmacological protection of retinal pigmented epithelial cells by sulindac
involves PPAR-alpha" in the prestigious Proceedings of the National Academy of Sciences.
2466 ) , the court held that a generic manufacturer cannot be held liable for inadequate warnings if its labeling faithfully tracks that of the proprietary drug--in this case, the nonsteroidal anti-inflammatory drug sulindac
In late June, the Supreme Court, however, cleared the manufacturer of sulindac
over Ms Bartlett's injuries such as full-body burns and blindness, in effect reversing a jury verdict that granted the woman $21 million damages.
Therapeutic potential of sulindac
hydroxamic acid against human pancreatic and colonic cancer cells.
Mice consuming the cherry diet, anthocyanins, or cyanidin had significantly fewer and smaller cecal adenomas than mice consuming the control diet or sulindac
, celecoxib), vitamins and their derivatives (e.
WHEN KAREN BARTLETT SUFFERED severe burns and near-blindness after taking the generic anti-inflammatory drug sulindac
, she sued the maker, Mutual Pharmaceutical Co.
Success rates of between 37-57% have been reported with indomethacin and sulindac
Synergistic effects of (-)-epigallocatechin gallate with (-)- epicatechin, sulindac
In some instances, COX-2 inhibitors such as celecoxib and the less commonly used NSAID sulindac
, have been associated with a cholestatic injury.
Combination treatment with arsenic trioxide and sulindac
enhances apoptotic cell death in lung cancer cells via activation of oxidative stress and mitogen-activated protein kinases.