synthase


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syn·thase

 (sĭn′thās′, -thāz′)

synthase

(ˈsɪnθeɪz)
n
an enzyme that catalyses a process of synthesis
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References in periodicals archive ?
Velours, "The modification of the conserved GXXXG motif of the membrane-spanning segment of subunit g destabilizes the supramolecular species of yeast ATP synthase," The Journal of Biological Chemistry, vol.
Novel antifungal drug candidate of SCY-078, SCYNEXIS, is an oral glucan synthase inhibitor and is also in development as oral as well as intravenous formulations for the treatment of invasive and life-threatening fungal infections.
Specifically, the team found that blocking the action of the mitochondrial ATP synthase enzyme stalled egg cell development from stem cells in experiments in fruit flies, one of the main organisms used to study cell biology.
NO can be synthesized from L-arginine using three different nitric oxide synthase (NOS) isoforms, two of which are constitutive, endothelial (eNOS, NOSIII) and neuronal (nNOS, NOSI) iso forms, manifested under physiological conditions, while the third isoform, inducible nitric oxide synthase (iNOS, NOSII), is biosynthesised only after the stimulation by a variety of stressors and cytokines [3].
Sucrose phosphate synthase (SPS) is considered to be a primary regulator for controlling biosynthesis and accumulation of sucrose and plays an important role in translocation and distribution of photoassimilate in higher plant.
But a new compound, called a CTP synthase inhibitor, may help solve that problem.
PLC-induced dilatative adaptation, and growth was found associated with increased inducible nitric oxide synthase and reduced arterial vascular endothelial growth factor and intracellular adhesion molecule-1 expression.
NO is a transmitter molecule synthesised from its precursor L-arginine by nitric oxide synthase (NOS).
Two plasmids, pTZPHAC1 and pTZPHAC2, which contained the PHA synthase genes (phaC1 and phaC2) from P.
The activation of endothelial nitric oxide synthase (eNOS) has been shown to contribute to its vasodilating properties.
Polymorphisms that have been shown to influence the BP response to diuretics have been identified in candidate genes encoding alpha-adducin (ADD1), subunits of G-proteins (GNB3 and GNAS1), endothelial nitric oxide synthase (eNOS), and ACE (12-14).
Objective: In this study, we investigated whether methionine synthase (MTR) A2756G and methionine synthase reductase (MTRR) A66G gene polymorphisms were related with infertility.