telangiectasia


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Related to telangiectasia: ataxia telangiectasia

tel·an·gi·ec·ta·sia

 (tĕl-ăn′jē-ĕk-tā′zhə) also tel·an·gi·ec·ta·sis (-ĕk′tə-sĭs)
n.
Chronic dilation of groups of capillaries causing elevated dark red blotches on the skin.

[New Latin : tel(o)- + Greek angeion, vessel; see angio- + Greek ektasis, expansion (from ekteinein, to stretch out : ek-, ex, ex- + teinein, to stretch; see ten- in Indo-European roots).]

tel·an′gi·ec·tat′ic (-tăt′ĭk) adj.

tel•an•gi•ec•ta•sis

(tɛlˌæn dʒiˈɛk tə sɪs)

n., pl. -ses (-ˌsiz)
chronic dilatation of the capillaries and other small blood vessels.
[1825–35; tel-2 + angi(o)- + Greek éktasis extension = ekta-, variant s. of ekteínein to stretch out (ek- ec- + teínein to stretch) + -sis -sis]
tel•an`gi•ec•tat′ic (-ˈtæt ɪk) adj.
Translations

tel·an·gi·ec·ta·si·a

n. telangiectasia, telangiectasis, condición causada por dilatación de los vasos capilares y arteriolas que puede formar un angioma.
hereditary hemorrhagic ______ hemorrágica
hereditaria;
lympathic ______ linfática.

telangiectasia

n telangiectasia; hereditary hemorrhagic — telangiectasia hemorrágica hereditaria
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Hereditary hemorrhagic telangiectasia (HHT) is a rare, autosomal dominant disorder characterized by recurrent epistaxis, telangiectasias, and multiorgan vascular dysplasia.
Hereditary hemorrhagic telangiectasia (HHT) is an autosomal dominant systemic disorder of angiogenesis characterized by vascular malformations in mucocutaneous tissues, visceral organs, and the central nervous system (1).
Idiopathic macular telangiectasia type 2 (MacTel 2 or perifoveal telangiectasia) is an acquired vascular disease of the macula primarily involving neural and glial cell degeneration and loss.
KS is associated with acral skin blistering inducible by trauma, mucosal inflammation, photosensitivity, progressive pigmentation, telangiectasia, and skin atrophy (Poikiloderma).
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Telangiectasia indicates dilated small vessels occurring on the upper part of body and face due to inflammatory disease, radiotherapy, long-term exposure to the sun, and chronic alcoholism (3).
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The acquired ataxia telangiectasia and Rad3-related protein kinase inhibitor program covers two clinical-stage compounds, VX-970 and VX-803.