aminoglycoside

(redirected from Aminoglycoside antibiotics)
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Related to Aminoglycoside antibiotics: gentamicin, Macrolide antibiotics

a·mi·no·gly·co·side

 (ə-mē′nō-glī′kə-sīd′, ăm′ə-)
n.
Any of a group of broad-spectrum antibiotics, such as streptomycin, derived from species of Streptomyces or Micromonospora bacteria and used to treat infections caused by gram-negative bacteria.
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In animal studies, ORC-13661 provided highly significant protection of hearing in rats exposed to high doses of aminoglycoside antibiotics, a common cause of human hearing loss.
Under the company's animal studies, ORC-13661 provided highly significant protection of hearing in rats exposed to high doses of aminoglycoside antibiotics, a common cause of human hearing loss.
Gentamicin Injection belongs to a group of medicines known as aminoglycoside antibiotics, which work by preventing bacteria from growing and by killing them.
The parameters for positive results of the tetracycline antibiotics tested are: oxytetracycline (10 [micro]g/kg), tetracycline (10 [micro]g/kg), chlortetracycline (10 [micro]g/kg), and doxycycline (20 [micro]g/kg); the parameters for positive results of the aminoglycoside antibiotics tested are: dihydrostreptomycin (10 [micro]g/ kg), gentamicin (50 [micro]g/kg), hygromycin (20 [micro]g/kg), kanamycin (20 [micro]g/kg), neomycin (20 [micro]g/kg), paromomycin (25 [micro]g/kg), spectinomycin (50 [micro]g/kg), and streptomycin (20 pg/ kg).
These enzymes possess the ability to reduce the synergetic effect of aminoglycoside antibiotics on the cell wall of bacteria.
Aminoglycoside antibiotics such as streptomycin, gentamycin and kanamycin are widely used to treat a wide spectrum of bacteria in China.
The importance of this gene is elimination of synergistic effects between penicillin and glycopeptide or aminoglycoside antibiotics by encoding a bi functional enzyme (6,7).
The susceptibility to the aminoglycoside antibiotics was less when compared with the studies done by Jyothi et al.
sup][8],[9],[17] This may explain the relatively better activity of isepamicin in comparison to other aminoglycoside antibiotics.
The resistance of clinical isolates to Aminoglycoside antibiotics varies with the specific drug the microorganism its mechanism of resistance the geographic area and many other factors.
As the Aminoglycoside antibiotics resistance could be caused by the enzymatic inactivation or efflux pumps overexpression (Nikaido 2009), resistance reversal effects of the 3-Benzylchroman derivative/Aminoglycoside combinations might as well be ascribed to inhibition against the inactivation or overexpression by 3-Benzylchroman derivatives, which should be investigated in the future study.