Cancer cells

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cells once believed to be peculiar to cancers, but now know to be epithelial cells differing in no respect from those found elsewhere in the body, and distinguished only by peculiarity of location and grouping.

See also: Cancer

Webster's Revised Unabridged Dictionary, published 1913 by G. & C. Merriam Co.
References in periodicals archive ?
Professor of Oral Pathology Tuula Salo investigates oral cancer and especially the interaction between cancer cells and their surrounding tissue.
A major "don't eat me" signal which cancer cells appear to use to stop the immune system from attacking them has been discovered by US researchers.
The study explores this communication between precancerous and cancer cells in the context of an enzyme called PI3K.
According to Cancer Research UK, cancer can come back if treatment failed to get rid of all the cancer cells. In such instances, the cells left behind may grow into a new tumor.
Jou explained that TGF-[beta]1 is a double-edged sword because in normal cells it controls cell proliferation and induces apoptosis, however, once the human body suffers from cancer, a high expression of PSPC1 will convert the function of TGF-[beta]1 to promote the survival and growth of cancer cells.
A Japanese cancer researcher has praised the ongoing human body-centric anime 'Cells at Work!' for accurately portraying how the immune system attacks cancer cells.
'Bad cholesterol (low-density lipoprotein or LDL) controls the trafficking of tiny vessels which also contain these integrins, and this has huge effects on the ability of cancer cells to move and spread throughout the body, ' said Thomas Grewal, an associate professor from University of Sydney.
The evidence doesn't come from tests in patients, but on cancer cells.
The front-line chemotherapy drug paclitaxel sets off a variety of molecular-level changes that allow breast cancer cells to escape from the tumor.
The research was designed to determine how inhibition of platelet activation through the use of aspirin might affect the proliferation of colon and pancreatic cancer cells.
As we well know, the "Warburg effect" is the main theory of the energy metabolism in cancer cells. In contrast to normal differentiated cells, which rely primarily on mitochondrial oxidative phosphorylation to generate the energy needed for cellular processes, most cancer cells instead rely on aerobic glycolysis.