cardiac muscle

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Related to Cardiac myocytes: cardiac hypertrophy, Cardiomyocytes

cardiac muscle

n.
The specialized striated muscle tissue of the heart; the myocardium.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

car′diac mus`cle


n.
1. a specialized form of striated muscle in the hearts of vertebrates.
2. the myocardium.
[1900–05]
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.

cardiac muscle

Involuntary striated muscle, found only in the heart. See muscle.
Dictionary of Unfamiliar Words by Diagram Group Copyright © 2008 by Diagram Visual Information Limited
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.cardiac muscle - the muscle tissue of the heartcardiac muscle - the muscle tissue of the heart; adapted to continued rhythmic contraction
muscular tissue, muscle - animal tissue consisting predominantly of contractile cells
ticker, heart, pump - the hollow muscular organ located behind the sternum and between the lungs; its rhythmic contractions move the blood through the body; "he stood still, his heart thumping wildly"
cardiac pacemaker, SA node, sinoatrial node, pacemaker - a specialized bit of heart tissue that controls the heartbeat
papillary muscle - any of several muscles associated with the atrioventricular valves; "the papillary muscles contract during systole to prevent regurgitation of blood into the atria"
atrioventricular bundle, atrioventricular trunk, bundle of His, truncus atrioventricularis - a bundle of modified heart muscle that transmits the cardiac impulse from the atrioventricular node to the ventricles causing them to contract
atrioventricular node - a node of specialized heart muscle located in the septal wall of the right atrium; receives impulses from the sinoatrial node and transmits them to atrioventricular bundle
myocardium - the middle muscular layer of the heart wall
Purkinje fiber - a specialized cardiac muscle fiber that is part of the Purkinje network
Purkinje network, Purkinje's system, Purkinje's tissue - a network of Purkinje fibers that carry the cardiac impulse from the atrioventricular node to the ventricles of the heart and causes them to contract
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.
References in periodicals archive ?
When a cAMP analogue or a PDE3I was administered to cultured cardiac myocytes transfected with promoter-reporter constructs containing one of the alleles, PDE3A transcription increased with the deletion, or DEL, variant.
Immunofluorescence labeling was used to measure the cell surface area, and 3H-leucine incorporation was used to detect the synthesis of total protein in neonatal rat cardiac myocytes (NRCMs) with the overexpression of miR-24.
Nitric oxide is produced from the cardiac endothelial cells as well as the cardiac myocytes in the heart and regulates heart functions.
The virus could directly damage cardiac myocytes to produce fibrosis, the virus could trigger cardiac inflammation, and the infected person could have an increased susceptibility to infection by a pathogen know to potentially cause cardiac damage and myocarditis such as coxsackievirus.
It is caused by the metabolic imbalance involving elevated energy requirements and deficient oxygen supply to the cardiac myocytes, ultimately leading to myocardial necrosis [2].
If oxygen demand outstrips supply, the cardiac myocytes experience injury, called myocardial infarction (MI); this commonly manifests as the release of intracellular cardiac enzymes such as troponin.
Cardiac troponin is a member of a complex group of proteins that modulate the calcium-mediated interaction between actin and myosin within the cardiac myocytes. (1) In acute myocardial infarction, cTn levels rise within 3 to 4 hours after the occurrence of clinical symptoms, reaching a peak at 12 to 16 hours, and can remain elevated for up to 9 to 14 days after myocardial infarction.
[5] demonstrated an increased density of [I.sub.NaL] in cardiac myocytes isolated from patients with HCM.
Mechanism of the prolongation and clinical manifestations are due to alterations in the rectifying potassium and sodium channels in cardiac myocytes. This may be directly due to toxicity of the channel, alterations in electrolytes, or congenital mutations of the channels themselves [9, 10].
Jongsma, "Differences in gap junction channels between cardiac myocytes, fibroblasts, and heterologous pairs," American Journal of Physiology-Cell Physiology, vol.
Specifically, TGF-[beta]1 induces expression levels of extracellular matrix (ECM) constituents by cardiac fibroblasts (i.e., fibrillar collagen, fibronectin, and proteoglycans), self-amplifies its own expression in both cardiac myocytes and fibroblast [53, 54], and stimulates the proliferation of fibroblasts and their phenotypic conversion to myofibroblasts [55, 56].