carnitine

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Related to Carnitine deficiency: L-carnitine

car·ni·tine

 (kär′nĭ-tēn′)
n.
A compound that functions in fatty acid metabolism by transporting fatty acids into mitochondria for energy production. It is naturally produced in the body and is present in many animal products, especially red meat. It is also sold as a dietary supplement for its purported health benefits.

[German Karnitin, from Karnin, a basic substance derived from meat, from Latin carō, carn-, flesh; see carnal.]

carnitine

(ˈkɑːnɪˌtaɪn)
n
a white betaine, C7H15NO3, found in the liver and required for transporting fatty acids from the cytosol into the mitochondria
Translations

carnitine

n carnitina
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References in periodicals archive ?
Hyperammonemia and carnitine deficiency treated with sodium valproate in psychiatric setting, hit Med J.
In this case, the child was supplemented with L-carnitine to correct the secondary carnitine deficiency and has remained asymptomatic with normal growth and development.
He brought carnitine into the United States and guided it through our entire system to obtain FDA approval which now saves the lives of children with the previously fatal disease, Primary Carnitine Deficiency.
Our hypothesis was that individuals with FMF would have lower FC levels than their healthy age and sex matched controls due to increased need of carnitine because of recurrent auto-inflammation; however, neither carnitine deficiency nor decreased FC levels compared to the healthy controls were detected in the study.
In our patient's case, cardiomyopathy secondary to carnitine deficiency from poor oral intake was the likely etiology.
Raskind and El-Chaar extensively reviewed the pathophysiology and significance of VPA-induced carnitine deficiency and recommended carnitine supplementation during VPA therapy.
Chen, "Primary carnitine deficiency and cardiomyopathy" Korean Circulation Journal, vol.
Transient infantile hypertriglyceridemia with associated hyperglycinemia and secondary carnitine deficiency.
16] primary carnitine deficiency are caused by improper energetic balance due to genetically determined flaws in fatty acid metabolism.
Therefore, carnitine biosynthesis and renal conservation mechanisms are generally implicated and adequate to prevent overt carnitine deficiency in individuals who self-select diets that are low in carnitine (42).
Carnitine transport by organic cation transporters and systemic carnitine deficiency.

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