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n. mielinólisis, enfermedad que destruye la mielina alrededor de ciertas fibras nerviosas;
acute ______ aguda;
___ transverse___ transversa.
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WD of MCPs can also occur after pontine hemorrhage and central pontine myelinolysis, not only pons infarction.
There is no risk of central pontine myelinolysis with this therapy in the context of EAHE.
alcohol use disorder [AUD]) from the various clinically diagnosable consequences of chronic alcohol consumption, including Wernicke's encephalopathy (WE), Korsakoff's syndrome (KS), hepatic encephalopathy (HE), central pontine myelinolysis (CPM), alcoholic cerebellar degeneration (ACD), alcohol-related dementia (ARD), and Marchiafava-Bignami disease (MBD).
7% 5,290 CNS-LS: Center for Neurological Study-Liability Scale; PBA: pseudobulbar affect Source: References 1,12 Table 3 Other disease states and injuries that have been associated with PBA Acute demyelinating encephalomyelitis Central pontine myelinolysis Cerebral aneurysms Cerebral arteriovenous malformations Cerebral palsy Cerebral tumors Chemotherapy Corticobasal degeneration Deep brain stimulation Frontotemporal dementia Herpes encephalitis Huntington's disease Lipid storage diseases Neurosyphilis Normal pressure hydrocephalus Primary lateral sclerosis Progressive supranuclear palsy Wilson's disease PBA: pseudobulbar affect Source: References 12-14
Differential diagnosis of central pontine myelinolysis includes infarct, metastasis, glioma, multiple sclerosis, encephalitis, radiotherapy and chemotherapy.
Catatonia was first described by Karl Kahlbaum in 1874 and has as an aetiology neurological disorders (encephalopaties, tumours, degenerative, central pontine myelinolysis, locked-in syndrome), psychiatry (hysteria, neuroleptic malignant syndrome, schizophrenia, psychosis, drug withdrawl), infectious diseases (AIDS, malaria, neurosyphilis), metabolic ones (Addison's, diabetic ketoacidosis, hepatic or renal failure, poisoning), but also inherited neurometabolic disorders such as homocystinuria, coproporphyria, Tay-Sachs disease.
Slow correction of serum sodium levels is necessary to avoid central pontine myelinolysis.
Hypernatremia is hazardous during liver transplantation, particularly in patients with low serum sodium levels who are at risk of central pontine myelinolysis due to rapid correction of hyponatremia.
3] Hyponatremia itself may cause central pontine myelinolysis, and may be an independent contributing factor to morbidity.
In contrast, clinical manifestations that primarily present as the coexistence of distal renal tubular acidosis and central pontine myelinolysis (CPM) are very rare in pSS.
Central pontine myelinolysis in a previously healthy 4-year-old child with acute rotavirus gastroenteritis.
The possibility of central pontine myelinolysis was also entertained, but no history of hyponatremia was found.
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