DTh

(redirected from Delayed-Type Hypersensitivity)
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D.Th.

or D.Theol.,

Doctor of Theology.
References in periodicals archive ?
One theory was based on the idea of a cell-mediated immune reaction, while another one sustained a delayed-type hypersensitivity reaction.
EN is a delayed-type hypersensitivity reaction, causing inflammation on the fat (panniculitis) most commonly on the shins, but it can also occur on the arms, face, neck, and thighs.
Indeed, the maculopapular skin eruption, associated with the administration of amoxicillin in the course of infectious mononucleosis, is considered to represent a delayed-type hypersensitivity reaction (type IV allergic reaction).
EN is a delayed-type hypersensitivity reaction, causing inflammation of the fat (panniculitis) most commonly on the shins, but it can also occur on the arms, face, neck, and thighs.
DTH: Delayed-type hypersensitivity Table 3: Results of bacteriological examination of challenged mice Mice groups Organ subjected to bacteriological examination Spleen (%) Liver (%) Lung (%) Kidney (%) Positive mice (%) I 30 10 20 20 30 II 30 0 10 0 30 III 10 0 20 10 10 IV 10 0 0 0 10 V 100 70 40 60 100 VI 100 80 60 40 100
The skin tests involving a prick or intradermal testing are considered to be crucial tools for evaluating drug hypersensitivity reactions, including IgE-mediated or delayed-type hypersensitivity, in both the European and American guidelines [22, 189-191].
[11] Mast cells have received little attention in leprosy, but evidence linking them with delayed-type hypersensitivity reactions raises the possibility that they may be of some importance in leprosy, in both the nonreactional and reactional states.
Several studies have shown that GOS supplementation enhances delayed-type hypersensitivity response in murine influenza vaccination model (Vos et al., 2006), natural killer cells activity and anti-inflammatory cytokines in human beings (Vulevic et al., 2008) and mice (Gopalakrishnan et al., 2012), and decreases pro-inflammatory cytokines production in humans (Vulevic et al., 2008).
ACD is a delayed-type hypersensitivity reaction in which a foreign substance comes into contact with the skin and is linked to skin proteins forming an antigen complex that leads to sensitization.
Platelets that have a role in delayed-type hypersensitivity are included in the complex etiopathogenesis of contact dermatitis.
It involves measuring induration caused by a delayed-type hypersensitivity reaction to Mycobacterium tuberculosis (Mtb) 2 or 3 days after injecting the reagent into the skin.