Hyperoxide


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Related to Hyperoxide: superoxide

Hy`per`ox´ide


n.1.(Chem.) A compound having a relatively large percentage of oxygen; a peroxide.
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Among them, the chemical oxygen supply commonly includes water-electrolyzed, potassium hyperoxide, peroxide, and oxygen candle.
In contrast, the composite with UV stabilizer had a lower rate of degradation than pure PP because the UV stabilizer inhibits the formation of hyperoxide group and ketone group, which are photoinitiators of PP, through the reprocessing of nitroxyl radicals [14,15].
Hyperoxide anions were produced from molecular oxygen in the presence of mercaptoethanol EDTA and Mn[Cl.sub.2].
ROS originate from the incomplete reduction of molecular oxygen resulting in the accumulation of oxidants like hydrogen peroxide ([H.sub.2][O.sub.2]) or free radicals like the hyperoxide (superoxide) anion ([O.sub.2.sup.*-]).
Loss or oxidation of B[H.sub.4] to 7,8-dihydrobiopterin (BH2) is associated with NOS uncoupling that results in the production of vasoconstrictory superoxide anion ([O.sub.2.sup.-], hyperoxide anion) at the expense of vasodilatory NO [34].
Acridine orange solution was prepared with 10 mL of stock solution, 40 mL of 0.1 M citric acid and 2.5 mL of 0.3 M diso-dium hyperoxide. The stock solution is prepared using 1000 mL distilled water and 1 g acridine orange hemi-zinc chloride (Sigma-Aldrich) and was stored at 4[degrees]C in the dark.
He offers "Manganerz, strahliges grau [= radiating gray], Ilefeld, Ilmenau, 15 kr.-10 fl." And "Mangan, Hyperoxide, Ilefeld, Ilmenau.
Serum-free culture (48 h), TNF[alpha] (40 ng/ml, 24 h) treatment, or hyperoxide ([H.sub.2][0.sub.2], 100 [micro]M, 24 h) stimulation induced apoptotic damage in myocytes, by about 3- to 5-fold increases in the number of apoptotic cells and ~30% reduction of the density of survival cells.
Both pAkt and caspase-3 protein expressions in these cells were increased by hyperoxide ([H.sub.2][0.sub.2]) stimulation and no further changes of these protein expressions were found in CP-treated cells under such condition.
The leading mechanism of the pathogenesis, which causes these damages of the cell membrane structure, is the activation of the lipid peroxidation which leads to the disturbance of balance between the formation and destruction of the hyperoxides and excessive accumulation of the free radicals which are toxic for the cells [15].