erg

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erg 1

 (ûrg)
n.
The centimeter-gram-second unit of energy or work equal to the work done by a force of one dyne acting over a distance of one centimeter.

[From Greek ergon, work; see werg- in Indo-European roots.]

erg 2

 (ûrg)
n.
An extensive area of desert covered with shifting sand dunes.

[Colloquial Arabic (Maghrebi dialect) 'arq, 'arg, from Arabic 'irq, vein, small mountain, thin tract of sand extending along the ground (in reference to the veinlike appearance of the dunes); see ʕrq in Semitic roots.]

erg

(ɜːɡ)
n
(Units) the cgs unit of work or energy. 1 erg is equivalent to 10–7 joule
[C19: from Greek ergon work]

erg

(ɜːɡ)
n, pl ergs or areg
(Physical Geography) an area of shifting sand dunes in a desert, esp the Sahara
[C19: from Arabic `irj]

erg

(ɜːɡ)
n
(Rowing) informal short for ergometer2

erg

(ɜrg)

n.
the centimeter-gram-second unit of work or energy, equal to the work done by a force of one dyne when its point of application moves through a distance of one centimeter in the direction of the force; 10−7 joule.
[1870–75; < Greek érgon work]

erg-

var. of ergo- 1 before a vowel: ergodic.

erg

(ûrg)
A unit used to measure energy or work, equal to the force of one dyne over a distance of one centimeter. This unit has been mostly replaced by the joule.

erg

1. An area in a desert where there are shifting sand dunes, for example in the Sahara.
2. A unit of energy equal to the energy produced by a force of 1 dyne acting through a distance of 1 cm. Replaced in the inernational system by the joule (J): 1 erg = 10-7 J.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.erg - a cgs unit of work or energy; the work done by a force of one dyne acting over a distance of one centimeter
energy unit, heat unit, work unit - a unit of measurement for work
J, joule, watt second - a unit of electrical energy equal to the work done when a current of one ampere passes through a resistance of one ohm for one second
Translations
エルグ

erg

[ɜːg] Nergio m, erg m

erg

nErg nt
References in periodicals archive ?
5% DSC2 Desmocollin 2 <2% JUP Junction plakoglobin N/A LQTS KCNQ1 Potassium voltage-gated 40%-55% channel subfamily Q member 1 KCNH2 Potassium voltage-gated 35%-45% channel subfamily H member 2 SCN5A Sodium voltage-gated 2%-8% channel alpha subunit 5 Brugada SCN5A Sodium voltage-gated channel alpha subunit 5 CPVT RYR2 Ryanodine receptor 2 50% (cardiac) CASQ2 Calsequestrin 2 (cardiac N/A muscle) TRDN Triadin N/A CALM1 Calmodulin 1 (phosphorylase N/A kinase, delta) KCNJ2 Potassium voltage-gated N/A channel subfamily J member 2 FH LDLR Low density lipoprotein 70%-80% receptor APOB Apolipoprotein B 2%-7% PCSK9 Proprotein convertase <1% subtilisin/ kexin type 9 ICC Gene References HCM MYBPC3 Alfares et al.
HTR2C, CHRM2, ADRA2B, HTR6, HTR5A, ADRA2C, HTR7, DRD2, and DRD4; SLC6A4, ADRA1A, HTR1A, ADRA1B, HTR2A, and DRD1; OPRL1, CHRM5, CHRM1, ADRB1, ADRA2A, and HRH1; and CHRM4, CHRM3, KCNH2, SLC6A4, CYP2D6, SCN1A: four clusters of neuronal GPCRs (G-proteincoupled receptors: adrenergic, cholinergic, histamine, and serotonin receptors).
Patient-specific iPSCs carrying different mutations in KCNH2 that encodes alpha-subunit of [I.
Gene mutations in KCNQ1, KCNH2 and SCN5A account for 90% to 95% cases of long QT syndrome.
While molecular autopsies involving the 4 major cardiac channelopathy genes (KCNQ1 [LQT1], KCNH2 [LQT2], SCN5A [LQT3, BrS1], and RYR2 [CPVT1]) have implicated LQTS, CPVT, and BrS as the underlying pathogenetic basis for an estimated 25% to 30% of SUD cases, (9-11) to date there are at least 28 channelopathy-susceptibility genes.
In the case at hand, the child had mutation in the SCN5A gene, which encodes a sodium channel, and a common polymorphism in the KCNH2 gene, which encodes a potassium channel.
The comprehensive triple-testing for the KCNQ 1, KCNH2, and SCN5A genes routinely recommended for long QT syndrome has a diagnostic yield of 80% and a signal-to-noise ratio of 19:1.
The majority of mutations have been identified in LQT1 (40%-55%), LQT2 (35%-45%), and LQT3 (2%-8%), which represent the genes KCNQ1, KCNH2, and SCN5A, respectively.