Mendelian laws

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Mendelian laws

n. leyes de Mendel, principios de rasgos hereditarios y la sucesión de cómo estos pasan de una generación a otra a través de genes dominantes y recesivos.
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Gene defects can occur through non- or under-expression of a gene, or through expression of a nonfunctional form of a gene, in a form which is recessive in Mendelian genetics terms; that is, in the presence of a "good" gene copy, the defect is masked or corrected for.
For example, in 1910, Mendelian genetics appeared to present a complete explanation of heredity and was used to justify an appalling program of segregation, marriage restriction, and sexual sterilization.
For example, Mendelian genetics arose and was appropriated by eugenicists to falsely link complex personal attributes to heredity.
The disease phenotype is inherited with Mendelian genetics, so that it occurs much less often than the genes - it only occurs when a child inherits a mutant gene from each parent.
He uses a non-reductionist approach, and asks whether a few aspects of older developmental and Lamarkian biology are worth remembering, not as alternatives to Mendelian genetics but as gropings toward an understanding of non-genetic factors interacting with genetic ones.
Mendelian genetics, which back then was not fully accepted by the scientific community, would exert a great influence on Vavilov's ideas.
Those of you who remember Mendelian genetics from your schooldays will know that for a recessive characteristic to emerge it is necessary for both parents to be carriers.
However, the largest gains were observed in questions related to the use of a Punnett square to solve Mendelian genetics (Q23 and Q24) and to understanding the relationship between cellular functions and cell division (Q31).
Such traits, which defy the predictions of classical Mendelian genetics, are also found in mammals.
We might add a caveat here: epigenetic studies suggest that inheritance is more complex than Mendelian genetics and such complexity is beyond the scope of junior high school Biology.
Maternal obesity may alter pups' body-weight-controlling mechanisms in a way not described by traditional Mendelian genetics.
Mendelian genetics and lifestyle risk factors play only a limited role in most childhood tumors, so routine screening and risk reduction efforts aren't emphasized.