Significant mitochondrial edemas in the degradation blanks were also observed in 36[degrees]C group (Picture (2); 1 h, C), and the most serious degration of myofibrils
was found in 36[degrees]C HS group (Picture (3); 2 h).
This increased weight of carcass may be due to the protection of glycogen reservoirs due to the extra supplementation of CHO in the pectoris muscle which is needed in the multiplication of myofibrils
in this area.
The structural basis for such a contraction process is derived from the highly ordered lattice structure of myofibrils
of muscle fibers.
"Muscle is made up of protein strands or myofibrils
so eating quality protein is key when it comes to building muscle," she says.
Chapman (1978) described the tentacle musculature of the Tripedalia cystophora cubopolyp as smooth longitudinal epitheliomuscular cells but noted the presence of striated myofibrils
near the distal tip of the tentacles.
However, when the CHD4 protein is absent, non-cardiac muscle proteins are inappropriately produced in developing heart muscle cells, forming abnormal, hybrid myofibrils
that lack the functional properties of a normal heart.
(Mc Ardle et al.2010, Garrett and Kirkendall 2000) Along with muscle hypertrophy, the number of myofibrils
and mitochondria increase intensively, and the phosphagen system develops.
The morphology of the EDL muscle from the CTL group exhibited sarcoplasm with the presence of well-defined myofibrils
and an organized Z-line (Fig.
Cruz-Hofling & Rodrigues-Simioni (1998) observed a reduction in the number of synaptic vesicles following the morphological alterations in the myofibrils
and mitochondria using guanidine.
In every muscle cell there are so-called myofibrils
, which contract at the same time.
Lesions in WMD are mediated by oxidative damage to cell membranes, which allows extracellular calcium to flow into the damaged cells, causing activation of calcium-dependent proteases that then degrade myofibrils
and other cellular components, leading to myodegeneration and myonecrosis (COOPER & VALENTINE, 2016).
 Theoretically, V[O.sub.2]max is a point where oxygen cost of active myofibrils
under certain work reaches the intracellular physiometabolic leveling in establishing balance between oxygen volume required to oxidation metabolism and V[O.sub.2] consumed in mitochondrial chemical reactions so that increasing work intensity does not raise consumption oxygen of active myofibrils