There were no differences in the prevalence of elevated SUVRs in the anterior cingulate, posterior cingulate, precuneus, or the lateral temporal, parietal, or occipital lobes
(all P greater than .
The brain magnetic resonance imaging is the diagnostic gold standard showing edema in the white matter of bilateral temporal, parietal, and occipital lobes
for neuroradiologic characteristics, totally reversible.
Magnetic resonance imaging (MRI) of the brain confirmed severe periventricular cystic changes around the bodies of the lateral ventricles, and highlighted the presence of two focal lesions in the occipital lobes
, with a hypointense centre on T1 weighted and a hyperintense on T2 weighted, and fluid attenuation inversion recovery (FLAIR) images with a rim, mildly hyperintense on T1 and hypointense on T2.
E and F, Frontal and occipital lobes
with appropriate gyration and lateral ventricles, respectively.
Vasogenic edema has been demonstrated in MRI, typically in the subcortical white matter, partially symmetrically in the parietal and occipital lobes
and sometimes in the cortex (1).
Posterior reversible encephalopathy syndrome (PRES) or reversible posterior leukoencephalopathy syndrome was first described by Hinchey, in 1996, as a reversible syndrome manifested with an acute headache, consciousness impairment, seizures, and visual deficits, associated with white matter changes predominately affecting the posterior parietal and occipital lobes
of the brain but also involving the brainstem, cerebellum, and other cerebral areas [1-3].
Several magnetic resonance imaging (MRI) results revealed infarcts in the bilateral temporal lobes and the parietal and occipital lobes
, which were inconsistent with any vascular distribution [Figure 1].
A repeat CT scan of the brain (Figure 2) on day 5 demonstrated development of extensive white matter abnormality predominantly within the frontoparietal lobes of the cerebrum bilaterally with mild white matter abnormality within the occipital lobes
and left cerebellar hemisphere.
a) Cerebellar lobes lesions, (b) occipital lobes
and dorsal brain stem lesions, and (c) left thalamus and occipital lobes
Both visual cortices were hyperintense on axial T2-weighted (a) series and in T2 and flair weighted series posterior parasagittal pattern of increased signal intensity in both occipital lobes
(Figures 2(a) and 2(b)).
These regions include areas in the frontal and parietal lobes, which "organise" the process of visualisation, together with areas in the temporal and occipital lobes
, which represent the items we wish to call to the mind's eye, and give visualisation its "visual" feel.
A computed tomography (CT) scan of her head without contrast showed low-density, patchy areas in the subcortical regions of the parietal and occipital lobes
bilaterally (FIGURE 1, arrows), with relative sparing of the cortex.