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Numerous growth factors are present in platelets like PDGF, EGF, VEGF, IGF, HGF, which play a pivotal role in tissue healing22.
* Lipid Bilayer-based Carriers loaded with Polypeptide Growth Factors: sh-Oligopeptide-1 (EGF), -4(Thymosin-b4); sh-Polypeptide-1 (FGF2), -3(FGF7), -4(SCF), -5(TGFb3), -7(hGH), -9(VEGF), -10(FGF10), -11(FGFl), -15(TIMP2), -22(TGFbl), -28(PRL), -40(hPL), -59(PDGF), -71 (VIP), -78(HSP90A), -93(CTGF), -101 (Adiponectin)
In recent studies, the correlation between angiogenic growth factors such as VEGF, basic fibroblast growth factor, TGF-[beta], and PDGF and formation, and recurrence of pterygium has been demonstrated (45-47).
Vascular endothelial growth factor (VEGF)-A and platelet-derived growth factor (PDGF) play a central role in the pathogenesis of digital clubbing.
A hypoxic environment induces VEGF and PDGF cytokine activities that promote both fibrogenic and angiogenic responses (42).
To our knowledge, no study has systematically examined the effects of PM2.5 on human bronchial epithelial cells (HBECs) in vitro and the possible molecular mechanisms that regulate the expression of TGF-[beta]1 and PDGF. According to epidemiological studies, PM2.5 might regulate the expression of HMGB1-RAGE signaling intermediates and associated mechanisms in elderly men .
IHC tests used several primary antibodies: TGFbeta1, TGFbeta2, TGFbeta3, TNFalpha, PDGF BB, and FGF1; source, clones, specific pretreatments, and dilutions are specified in Table 1.
TGF-[sz] is the strongest profibrotic factor in EMT of peritoneal mesothelial cells (PMCs) and a key mediator in dialysis-related peritoneal fibrosis.[sup], Platelet-derived growth factor (PDGF) also induced partial EMT in vivo and stimulated human PMC proliferation in vitro .[sup],As TGF-[sz]R and PDGF receptors (PDGFR) are both glycosylated, and CF is their common posttranslational modification,[sup] we believed that blocking CF may simultaneously inhibit TGF-[sz] and PDGF signaling pathways, resulting in a synergistic protective effect in ameliorating EMT.
PRF exudates contain good amount of growth factors (transforming growth factor (TGF)-[beta]1, platelet-derived growth factor (PDGF)-[beta], and vascular endothelial growth factor (VEGF)), matrix glycoprotein's (fibronectin and vitronectin), and proteins specialized in increasing cell attachment to biomaterial impregnation, rinsing surgical sites, hydration of graft materials, and for storage of autologous grafts.
I present here data for the candidate of activation factors such as cytokine interferon (IFN)-[gamm] and platelet-derived growth factor (PDGF) for evoking microglial activation.
Many studies have confirmed that growth factors are necessary for healing for example, platelet-derived growth factor (PDGF) enhance the wound healing rate in acute wounds and even provide complete healing in chronic wounds [106-108].
Nilotinib inhibits the following kinases, in order of potency: Discoidin domain receptor 1 (DDR 1) > DDR-2 > Bcr-Abl > platelet-derived growth factor (PDGF) receptor-a/-b > KIT > colony stimulating factor 1 receptor.
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- PDGF A-chain
- PDGF receptor
- PDGF subunit A
- PDGF subunit B
- PDGF, B chain
- PDGFA-Associated Protein 1