BChE

(redirected from Pseudocholinesterase)
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Related to Pseudocholinesterase: atypical pseudocholinesterase

BChE

abbr.
Bachelor of Chemical Engineering

B.Ch.E.

Bachelor of Chemical Engineering.
References in periodicals archive ?
Pseudocholinesterase deficiency: a comprehensive review of genetic, acquired, and drug influences.
Low pseudocholinesterase (PChE), high creatinine (Cr), low Glasgow Coma Scale (GCS) scores and long hospitalization durations were all found to be poor prognostics in MV patients.
sup][1],[2] Cholinesterase can be divided into two categories: acetylcholinesterase (AChE) and pseudocholinesterase.
Patients with atypical variants of pseudocholinesterase with deficient enzymatic activity may require mechanical ventilation and close monitoring following administration of this muscle relaxant.
BuChE is also known as pseudocholinesterase as its capability to hydrolyse both ACh and BuCh at different frequencies (Forget et al.
1) Butyrylcholinesterase, also called pseudocholinesterase, has drawn growing interest in human medicine and is used as a target in Alzheimer's disease (16,17) and Parkinson disease treatment.
In this paper they describe four patients with pseudocholinesterase deficiency, all of whom demonstrated TOF ratio fade during the block, which they claimed demonstrated that the block had features of a non-depolarising block.
The patient's medical history revealed nothing of consequence, and the plasma uric acid level and other routine preoperative blood tests were within normal limits, except for a slightly elevated level of the plasma pseudocholinesterase enzyme activity.
Other diseases of connection to Sephardi origin include: Alpha-Thalassemia, Ataxia Telangiectasia, Corticosterone Methyloxidase Type II Deficiency, Costeff Optical Atrophy, Cystic Fibrosis (CF), Familial Creutzfeldt-Jakob Disease, Familial Tumoral Calcinosis (Normophosphatemic Type), Inclusion Body Myophy Type 28, Metachromic Leukodystrophy, Polyglandular Deficiency Syndrome, Pseudocholinesterase Deficiency, Spinal Muscular Athrophy (S.
Analysis of the human genome indicates that there are several forms of genetic variation, including nucleotide deletions, insertions, inherited deficiency of the enzyme pseudocholinesterase, numerous examples of polymorphisms in genes encoding drug-metabolising enzymes, drug transporters and drug targets (enzymes, receptors) have oligonucleotide repeats and single nucleotide polymorphisms (SNPs)-stable, discrete, single-nucleotide substitutions that occur in [greater than or equal to]1% of the population (lower-frequency variations are considered mutations--been described.
Metabolism may be slower in patients with genetically abnormal pseudocholinesterase.
The symptomatology of organophosphorous insecticides is that they irreversibly inhibit both cholinesterase and pseudocholinesterase activity.