NS

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NS

abbr.
1. or N/S New Style
2. Nova Scotia
3. nuclear ship

ns

abbr.
nanosecond
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

ns

abbreviation for
1. new series
2. not specified

NS

abbreviation for
1. New Style (method of reckoning dates)
2. not sufficient or not satisfactory
3. (Placename) (esp in postal addresses) Nova Scotia
4. (Nautical Terms) nuclear ship
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014

NS

Nova Scotia.

ns

or nsec,

nanosecond.

N.S.

Nova Scotia.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
References in periodicals archive ?
Netherton Syndrome is an autosomal recessive monogenic disease caused by mutations in the SPINK5 gene that encodes a serine peptidase inhibitor.
Netherton syndrome is caused by homozygous or compound heterozygous mutations in the SPINK5 gene, which encodes the serine protease inhibitor LEKTI (lymphoepithelial Kazal-type-related inhibitor), on chromosome 5q32 (2).
Wu et al., "Distinct Spink5 and Il-31 polymorphisms are associated with atopic eczema and nonatopic hand dermatitis in Taiwanese nursing population," Experimental Dermatology, vol.
Netherton syndrome is caused by germline mutations in the serine protease inhibitor of Kazal type 5 (SPINK5) gene located on chromosome 5q31-32 [2].
The proteolytic activity is dependent on pH in the SC and is regulated by a cocktail of protease inhibitors, including lymphoepithelial Kazal-type 5 serine protease inhibitor (LEKTI) encoded by the serine protease inhibitor Kazal-type 5 (SPINK5) [48].
FLG has several site-specific functions governed by the program of epidermal terminal differentiation in which a number of proteases are involved, such as corneum trypsin- and chymotrypsin-like enzymes (stratum corneum tryptic enzyme/kallikrein 5 [KLK5] and stratum corneum chymotryptic enzyme/KLK7),[sup][9],[10],[11] channel-activating serine protease1 (CAP1),[sup][12] matriptase (coded by ST14),[sup][12] and cysteine protease caspase-14[sup][13] and their key protease inhibitor lymphoepithelial Kazal-type-related inhibitor (LEKTI) (encoded by serine protease inhibitor Kazal-type 5 gene/ SPINK5 ).[sup][12],[13],[14] All of the proteases can enhance FLG degradation processing.
In addition, the mutations in the filaggrin gene and the SPINK5 (serine protease inhibitor kazal-type 5) gene are associated with defective epidermal differentiation and skin barrier formation [5].