Wernicke-Korsakoff syndrome


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Related to Wernicke-Korsakoff syndrome: Wernicke's encephalopathy, thiamine, pellagra, beriberi

Wer·nick·e-Kor·sa·koff syndrome

 (vĕr′nĭ-kē-kôr′sə-kôf′, -kŏf′, -nĭ-kə-)
n.
A neurological disorder usually associated with severe alcoholism and subsequent thiamine deficiency, marked by the signs of Wernicke's encephalopathy in the acute phase followed by the chronic memory loss and disorientation of Korsakoff syndrome.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.
References in periodicals archive ?
Although neither Wernicke nor Korsakoff noticed the relationship between these two entities (5), for today the syndrome is usually referred to under one name, Wernicke-Korsakoff syndrome (WKS).
He found secondary factors were neglect of clinical needs, carcinoma of the tongue and Wernicke-Korsakoff syndrome.
Thiamine deficiency causes Beri-Beri and Wernicke-Korsakoff syndrome [10,11].
Cook, "Wernicke-Korsakoff syndrome not related to alcohol use: a systematic review," Journal of Neurology, Neurosurgery & Psychiatry, vol.
Ultimately, the patient may develop infantile beriberi, shock, Wernicke-Korsakoff syndrome, or even death.
"TD is known to cause neuron impairments such as Wernicke-Korsakoff Syndrome. Although how exactly TD can cause neural impairments is unclear, it is well known that oxidation stress cause telomere shortening and, thus, it is possible that oxidation stress may also cause neuron death," explained Yamaki.
The Wernicke-korsakoff Syndrome and Related Disorders Due to Alcoholism and Malnutrition.
Wernicke-Korsakoff syndrome (WKS) is used to refer to the presence of both WE and KS because of the close relationship between the two disorders.
The study also concluded that the incidence of alcohol hallucinosis, Wernicke's encephalopathy and Wernicke-Korsakoff syndrome was significantly and consistently higher in patients with higher quantity of alcohol intake.
They first address specific conditions and basic mechanisms in dementia and cognitive decline, including Alzheimer's disease, amyotrophic lateral sclerosis, corticobasal degeneration, Creutzfeldt-Jakob disease, frontotemporal lobar degeneration, Gaucher disease, Huntington's disease, dementia with Lewy bodies, normal pressure hydrocephalus, Parkinson's disease, Pick's disease, posterior cortical atrophy, progressive supranuclear palsy, cognitive vascular impairment, Wernicke-Korsakoff syndrome, risk factors, malnutrition, the role of diet in chronic inflammation and innate immunity in Alzheimer's, pathophysiological mechanisms and nutrition, and body composition.