adipocyte


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Related to adipocyte: reticular fiber

ad·i·po·cyte

 (ăd′ə-pō-sīt′)
n.

adipocyte

(ˈædɪpəʊˌsaɪt)
n
(Physiology) a fat cell that accumulates and stores fats

fat′ cell`



n.
a cell in loose connective tissue that is specialized for the synthesis and storage of fat. Also called adipocyte.
[1910–15]
Translations
tuková buňka
adiposyyttirasvasolu

ad·i·po·cyte

n. adipocito, célula adiposa.
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References in periodicals archive ?
One branch was MSCs to adipocyte progenitors to pre-adipocytes.
where the effects of curcumin on preadipocyte to adipocyte differentiation was investigated revealed that dose-dependent curcumin reduced adipocyte differentiation.
In vivo AT is highly vascularized, whereby each adipocyte is in contact with at least one blood vessel (Silha et al., 2005).
Claussnitzer et al .[4] observed that FTO gene variant can disrupt AT-rich interactive domain 5B repressor binding; this disruption results in derepression of IRX3 during early adipocyte differentiation.
To test this hypothesis, in the present study we investigated the expression pattern of GPR43 during the differentiation of brown adipocyte precursor cells.
A reciprocal relationship between hematopoiesis and adipocyte differentiation in the bone marrow has long been observed in experiments and in the clinic [1].
While an underlying proinflammatory state increases D2 activity and can affect lymphocyte proliferation [23], no human study to date has assessed the relationship between adipocyte DIO2 expression and pro- and anti-inflammatory factors and its possible impact on glucose homeostasis.
Similar to previous literature regarding atrophic adipocyte mimickers [10], the fat cells in this case are smaller than normal mature adipocytes and show slight variations in size and shape.
AMPK suppresses the expression of C/EBP-[alpha], C/EBP-[beta], C/EBP-[delta], and PPAR-[gamma] [29], and AMPK activator AICAR suppresses lipid metabolism by regulating [beta]-oxidation-associated proteins during adipocyte differentiation [30].
KLF4, originally separated from the gastrointestinal tract, is one of the transcriptional regulation factors of adipocyte differentiation [6].
Adipocyte fetuin-A contributes to macrophage migration into adipose tissue and polarization of macrophages.