Also found in: Thesaurus, Medical, Encyclopedia, Wikipedia.


 (ăl-byo͞o′mə-no͝or′ē-ə, -nyo͝or-)
The presence of albumin in the urine, sometimes indicating kidney disease.

al·bu′mi·nu′ric (-no͝or′ĭk, -nyo͝or′-) adj.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.


(Pathology) pathol the presence of albumin in the urine. Also called: proteinuria
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014


(ælˌbyu məˈnʊər i ə, -ˈnyʊər-)

the presence of albumin in the urine.
al•bu`mi•nu′ric, adj.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.albuminuria - the presence of excessive protein (chiefly albumin but also globulin) in the urinealbuminuria - the presence of excessive protein (chiefly albumin but also globulin) in the urine; usually a symptom of kidney disorder
symptom - (medicine) any sensation or change in bodily function that is experienced by a patient and is associated with a particular disease
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.


n. albuminuria, presencia de proteína en la orina, esp. albúmina o globulina.
English-Spanish Medical Dictionary © Farlex 2012


n albuminuria
English-Spanish/Spanish-English Medical Dictionary Copyright © 2006 by The McGraw-Hill Companies, Inc. All rights reserved.
References in periodicals archive ?
Sjostrom, "Dapagliflozin reduces albuminuria in patients with diabetes and hypertension receiving renin-angiotensin blockers," Diabetes, Obesity & Metabolism, vol.
Many studies have shown that increased urinary protein and albuminuria are associated with increased risk of ESRD.
In a renal endpoints trial of patients at high cardiovascular risk, progression of albuminuria and the renal composite outcome (composed of sustained reduction in eGFR, the need for renal replacement therapy, or death from any cause) occurred less frequently in patients taking canagliflozin (Neal et al., 2017).
Albuminuria was defined as the ratio of urine albumin to creatinine: A1, <30 mg/g, A2, 30-300 mg/g, A3, >300 mg/g.
A 2011 meta-analysis of 5 RCTs (total 2975 patients) that compared ACE inhibitor therapy with placebo in diabetic patients without hypertension and albuminuria found that ACE inhibitors reduced the risk of new-onset microalbuminuria or macroalbuminuria by 18% (relative risk [RR]=0.82; 95% confidence interval [CI], 0.73-0.92).
Various traditional risk factors such as male gender, age, obesity, dyslipidemia, hypertension (HTN), poor glycemic control (high glycosylated hemoglobin (Hb[A.sub.1c])), albuminuria, smoking, and family history of CVD have been identified to be independent contributors for CVD.
Central Australian clinical databases were retrospectively audited for serum creatinine (sCR), albuminuria (ACR), glycated Hb (HbA1c), and eGFR for the five years preceding HD-start.
Laboratory findings are as follows: creatinine of 0.34mg/dl (estimated glomerular filtration rate by Schwartz formula of 183ml/min), albuminuria of 2.7 ug/min (reference value from 0 to 15), and proteinuria of 30 mg/24 hours (reference value < 150), with blood Lyso-Gb3 of 69.9 nmol/l (reference value < 1.2), normal electrocardiogram for their age, Doppler echocardiography with physiologic tricuspid and pulmonary regurgitation, normal brain magnetic resonance imaging, ophthalmological examination with slit lamp showing cornea verticillata in both eyes, and normal abdominal and renal ultrasound.
Thus, we set out to examine the increase in albuminuria and the involvement of podocytes in the kidney injury caused by experimental hyperuricemia in conjunction with the involvement of oxidative stress.
In most cases, recognition of the diabetic kidney disease (DKD) is based on results of tests such as albuminuria, creatininemia with the estimation of eGFR, or renal imaging.
In a randomized, controlled, multicenter trial (ADVANCE study), high albuminuria and low eGFR were proved to be independent risk factors for cardiovascular events among T2DM patients.[sup][5] Moreover, in another prospective cohort study in the United States, CKD was confirmed to be associated with an increase in stroke risk.[sup][6]
Significantly increased albuminuria was noted in patients with chronic kidney disease who continued to smoke after initiating ACE inhibitor therapy, based on results from a 5-year follow-up study of 108 patients who smoked cigarettes at study entry -25 of whom quit smoking within the first year of the study--and 108 patients who never smoked.