angiotensin II

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Noun1.angiotensin II - a potent vasopressor agent formed from angiotensin I
angiotensin, angiotonin, Hypertensin - any of several vasoconstrictor substances (trade name Hypertensin) that cause narrowing of blood vessels
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References in periodicals archive ?
The EC's approval is based on data from the ATHOS-3 (Angiotensin II for the Treatment of High-Output Shock) Phase 3 study, which established the safety and efficacy of GIAPREZA in adults with septic or other distributive shock.
Withholding versus continuing angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers before noncardiac surgery: an analysis of the Vascular Events in Noncardiac Surgery Patients Cohort Evaluation prospective cohort.
Although the rats' blood pressure levels in recovery remained normal, higher-than-normal blood pressure responses to angiotensin II may increase the risk of developing high blood pressure.
Another example of new codes in Table XWO is Giapreza, an FDA-approved synthetic human angiotensin II used to raise the critically low blood pressure in a patient with septic or other distributive shock, which can affect blood flow to vital organs.
[6.] Johren O, Dendorfer A, Dominiak P (2004) Cardiovascular and renal function of angiotensin II type-2 receptors.
Hence it is hypothesized that lisinopril mediate anti-inflammatory effect via reducing the action of angiotensin II.
RAS regulates the blood pressure by two receptors of angiotensin II receptor 1 (AT1R) and angiotensin II receptor 2 (AT2R) (6, 8).
The presentation, entitled "Outcomes in Patients with Acute Kidney Injury Receiving Angiotensin II for Vasodilatory Shock," will take place during The 23rd International Conference on Advances in Critical Care Nephrology - AKI & CRRT 2018, being held March 6-9.
However, recent evidence suggests a potential benefit of type 2 angiotensin II (Ang II) receptor (AT2R) stimulation for improving both lipotoxicity and hyperandrogenism.
Pressure overload and cardiac hypertrophy share common inducers (e.g., endothelin and angiotensin II) that activate downstream matrix metalloproteinases (MMPs, such as MMP2 and MMP7) and a disintegrin and metalloproteinases (ADAMs, such as ADAM12 and ADAM17) via activating Gq protein-coupled receptors [2, 5-7].
However, angiotensin II (Ang II) and other cytokines reduce the number and bioactivities of EPCs in patients [9-11].
A previous study in adult rats also revealed that myocyte stretching that mimics volume overload can induce angiotensin II synthesis and myocyte apoptosis, which could then be abolished by angiotensin II type I receptor blocker [6].

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