app

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APP

abbr.
amyloid precursor protein

app

 (ăp)
n.
A computer application.

[Short for application.]
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

app

(æp)
n
(Computer Science) computing (esp downloaded to a mobile device) short for application program
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014

app

(æp)
n.
Computers (informal). an application program; application software.
[1985–90]

app.

1. apparatus.
2. apparent.
3. appendix.
4. applied.
5. appointed.
6. approved.
7. approximate.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
References in periodicals archive ?
The cause of the disease is the accumulation of the toxic beta-amyloid peptide in brain tissues, which leads to oxidative stress, neuroinflammation, mitochondrial dysfunction, and ultimately - the death of nerve cells.
Buyukserin, "Fabrication of thioflavin-T-modified nanopillared SERS substrates for ultrasensitive beta-amyloid peptide detection," Journal of Raman Spectroscopy, vol.
Link, "Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans," Proceedings of the National Academy of Sciences of the United States of America, vol.
Davila-Rodriguez et al., "Beta-amyloid peptide in blood clots," in Proceedings of VI Meeting of the Physiol, pp.
Herron, "Inhibition of LTP in vivo by beta-amyloid peptide in different conformational states," Brain Research, vol.
Reduced VEGF levels within this microvascular fraction of the brain by impairing overall angiogenesis and possibly reducing blood-brain barrier permeability could alter the normal homeostatic balance of beta-amyloid peptide handling, leading to impaired efflux out of the CNS parenchymal compartment leading to net accumulation.
The researchers first created a virtual beta-amyloid peptide that only contained the last 12 amino acids of the entire Abeta42 protein.
Since then, these have been revealed as accumulations of two separate proteins in the brain: beta-amyloid peptide and tau proteins.
The SORL1 gene is a coding gene for a protein involved in the production of the beta-amyloid peptide. This protein is known to affect the functioning of the brain cells (see insert).
Two of the identified mutations are responsible for an under-expression of SORL1, resulting in an increase in the production of the beta-amyloid peptide. "The mutations observed on SORL1 seem to contribute to the development of early-onset Alzheimer's disease.
Disease-modifying strategies for the treatment of Alzheimer's disease targeted at modulating levels of the beta-amyloid peptide. In Biochem.