cobalamin


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co·bal·a·min

 (kō-băl′ə-mĭn) also co·bal·a·mine (-mēn′)

cobalamin

(kəʊˈbæləmɪn)
n
(Biochemistry) vitamin B12

vitamin B1


n.
[1920–25]

vitamin B2


n.
[1925–30]

vitamin B3


n.
[1975–80]

vitamin B6


n.
[1930–35]

vitamin B12


n.
a complex water-soluble solid, C63H88N14O14PCo, obtained from liver, milk, eggs, fish, oysters, and clams: a deficiency causes pernicious anemia and disorders of the nervous system. Also called cyanocobalamin, cobalamin, extrinsic factor.
[1945–50]

co·bal·a·min

(kō-băl′ə-mĭn)
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.cobalamin - a B vitamin that is used to treat pernicious anemiacobalamin - a B vitamin that is used to treat pernicious anemia
B complex, B vitamin, B-complex vitamin, vitamin B, vitamin B complex, B - originally thought to be a single vitamin but now separated into several B vitamins
References in periodicals archive ?
HTF MI released a new market study on Vitamin B12 (Cobalamin, Cyanocobalamin) Market with 100+ market data Tables, Pie Chat, Graphs & Figures spread through Pages and easy to understand detailed analysis.
Molecular mechanisms leading to three different phenotypes in the cblD defect of intracellular cobalamin metabolism.
Multiple linear regression was used to investigate the association of cobalamin with fasting blood sugar level after adjusting for gestational age and gravidity.
Neuropsychiatry disorders caused by cobalamin deficiency in the absence of anemia or macrocytosis.
Hyperhomocysteinemia and elevated methylmalonic acid indicate a high prevalence of cobalamin deficiency in Asian Indians.
Performance of the serum cobalamin assay for diagnosis of cobalamin deficiency.
The disorders of intracellular cobalamin metabolism result from deficient synthesis of AdoCbl and/or MeCbl derived from vitamin [B.sub.12].
Poor vitamin B12 (cobalamin) status is widespread in South Asia and has been linked to poor neurodevelopment in young children.
Atypical hemolytic uremic syndrome (aHUS) is a life-threatening disease characterised by thrombotic microangiopathy (TMA) that is mainly due to Shiga toxin-producing Escherichia coli infections and uncontrolled complement activation, although it may rarely be due to other causes, such as disorders of intracellular cobalamin (cbl) metabolism [1].
Patients with inflammatory bowel conditions often suffer from cobalamin deficiency [1].