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 (ə-trē′zhə, -zhē-ə)
1. The absence or closure of a normal body orifice or tubular passage such as the anus, intestine, or external ear canal.
2. The degeneration and resorption of one or more ovarian follicles before a state of maturity has been reached.

[New Latin atrēsia : Greek a-, not, without; see a-1 + Greek trēsis, perforation, orifice; see terə- in Indo-European roots.]

a·tre·sic (-zĭk, -sĭk) adj.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.


(əˈtriːʒɪə; -ʒə)
(Pathology) absence of or unnatural narrowing of a body channel
[C19: New Latin, from Greek atrētos not perforated]
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014


(əˈtri ʒə, -ʒi ə)

the absence, or failure to develop, of a normal body opening or duct, as the ear canal.
[1800–10; < Greek a- a-6 + três(is) perforation + -ia -ia]
a•tre′sic (-zɪk, -sɪk) a•tret•ic (əˈtrɛt ɪk) adj.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.atresia - an abnormal condition in which a normal opening or tube in the body (as the urethra) is closed or absent
abnormalcy, abnormality - an abnormal physical condition resulting from defective genes or developmental deficiencies
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.


n. atresia, cierre congénito anormal de una abertura o conducto del cuerpo.
English-Spanish Medical Dictionary © Farlex 2012
References in periodicals archive ?
Ovarian follicular atresia and postovulatory regression in mammals is mediated by apoptosis, which is a natural occurring cause of cellular death.
Previous studies reported that melatonin ensures a protective effect on premature ovarian insufficiency in ovarian and increasing ovarian follicles, decreasing follicular atresia [20-22].
They can also decrease breakdown in the ovaries of the eggs and follicles (follicular atresia), delaying the onset of natural menopause.
Incidences of follicular atresia increase late in the reproductive season when yolked follicles that did not complete yolk deposition were resorbed (Goldberg 1973), thus conserving energy reserves that may be utilized in the subsequent period of reproduction.
These compounds by binding to the active site of the progesterone-metabolizing enzyme prevent the transformation of progesterone to 3-a 5-a tetrahydroprogesterone; therefore, it may be the main cause of the progesterone hormone increase.[43],[44] Several studies have reported that the apigenin inhibits aromatase activity.[45],[46] Moreover, genistein is one of the phytoestrogen compounds of hydroalcoholic extract of chamomile that can impact estrogen signaling pathways because of binding to estrogen receptors.[47] Several studies have shown that genistein can inhibit steroidogenic enzymes and steroidogenesis, induce follicular atresia, and decrease oocyte maturation.[48],[49] Patel et al.
During the juvenile stage, the taiep rat showed a significant decrease in the follicular population, with a decrease in the primary follicles and an increase in the preantral follicles and follicular atresia. This may be a consequence of the increase in the plasma concentration of testosterone [29].
BMP15 is mitogenic for somatic cells and is known to induce granulosa cell proliferation and affect the choice of the dominant follicle as well as the formation of follicular atresia (12).
This study is the first to report that apoptotic miR-378 plays a regulatory role in bGCs, which will help to find a new way in the research of follicular atresia.
In another study histological observations of folliculogenesis and follicular atresia in immature female rats (0-35 days old) were studied; after day 30 numerous primary follicles and atretic follicles were observed in the ovaries, antrum grew larger in the follicles where oocyte disappeared and granulosa cell layer became thin.8
Proposed mechanisms include increased ovarian growth factors, decreased follicular atresia, and increased expression of FSH receptors.
Meantime, this can lead to abnormal proliferation or excessive apoptosis of follicular cells such as the high LH dependent theca cellular proliferation [67] and follicular atresia induced granulosa cell inactivation [68-70] in PCOS and depletion of oocytes pool in premature ovarian failure [71].

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