glucose

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Related to glucose-6-phosphatase: glucose-6-phosphate dehydrogenase

glu·cose

 (glo͞o′kōs′)
n.
1. A monosaccharide sugar, C6H12O6, that is used by living things to obtain energy through the process of aerobic respiration within cells. It is the principal circulating sugar in the blood of humans and other mammals.
2. A colorless to yellowish syrupy mixture of dextrose, maltose, and dextrins containing about 20 percent water, used in confectionery, alcoholic fermentation, tanning, and treating tobacco. Also called starch syrup.

[French, from Greek glukus, sweet.]

glucose

(ˈɡluːkəʊz; -kəʊs)
n
1. (Elements & Compounds) a white crystalline monosaccharide sugar that has several optically active forms, the most abundant being dextrose: a major energy source in metabolism. Formula: C6H12O6
2. (Elements & Compounds) a yellowish syrup (or, after desiccation, a solid) containing dextrose, maltose, and dextrin, obtained by incomplete hydrolysis of starch: used in confectionery, fermentation, etc
[C19: from French, from Greek gleukos sweet wine; related to Greek glukus sweet]
glucosic adj

glu•cose

(ˈglu koʊs)

n.
1. a simple sugar, C6H12O6, that is a product of photosynthesis and is the principal source of energy for all living organisms: concentrated in fruits and honey or readily obtainable from starch, other carbohydrates, or glycogen.
2. a syrup containing dextrose, maltose, and dextrine, obtained by the incomplete hydrolysis of starch.
[1830–40; < French < Greek glykýs sweet]
glu•cos′ic, adj.

glu·cose

(glo͞o′kōs′)
A crystalline sugar having the formula C6H12O6, found in plant and animal tissue and essential to the animal diet. It is transported by blood and lymph to all the cells of the body, where it is broken down to produce ATP, the main source of energy for cellular processes.

glucose

(or dextrose) A simple sugar: the form of carbohydrate absorbed from the alimentary canal, supplied by blood to the muscles and converted for storage to glycogen.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.glucose - a monosaccharide sugar that has several formsglucose - a monosaccharide sugar that has several forms; an important source of physiological energy
aldohexose - a monosaccharide sugar having six carbon atoms and an aldehyde group
glucosamine - an amino derivative of glucose that is a component of many polysaccharides
corn sugar - dextrose made by hydrolysis of cornstarch
dextroglucose, dextrose, grape sugar - an isomer of glucose that is found in honey and sweet fruits
blood glucose, blood sugar - glucose in the bloodstream
Translations
جلوكوزجلوكوز: سُكَّر عِنَب
glucosa
glukózahroznový cukr
glukosedruesukker
glukozo
glukoosi
glukoza
szõlõcukor
glúkósi
グルコースブドウ糖
글루코오스포도당
gliukozė
glikoze
glukózaglukoza
glukosblodsockerdextrosdruvsocker
กลูโคส
đường glucose

glucose

[ˈgluːkəʊs] Nglucosa f

glucose

[ˈgluːkəʊz ˈgluːkəʊs] nglucose m

glucose

nGlucose f, → Glukose f, → Traubenzucker m

glucose

[ˈgluːkəʊs] nglucosio

glucose

(ˈgluːkous) noun
a kind of sugar found in the juice of fruit.

glucose

جلوكوز glukóza glukose Glukose γλυκόζη glucosa glukoosi glucose glukoza glucosio グルコース 글루코오스 glucose glukose glukoza glicose глюкоза glukos กลูโคส glükoz đường glucose 葡萄糖

glu·cose

n. glucosa, dextrosa, azúcar de fruta, fuente principal de energía en organismos vivos;
blood level of ___nivel de ___ en la sangre;
___, tolerance testprueba de tolerancia a la ___.

glucose

n glucosa
References in periodicals archive ?
11[beta]-HSD-1 is mainly found in the tissues such as liver, adipose, skeletal muscle, brain and immune system, over expression or increased activity of 11[beta]-HSD-1 leads to increase in cortisol levels in the body: especially in liver increased activity of 11[beta]-HSD-1 leads to increase in gluconeogenesis through the activation of hepatic phospho-enolpyruvate carboxykinase (PEPCK) and glucose-6-phosphatase activity (Radahmadi et at., 2006; Park et al., 2011) and that leads to hyperglycemia.
The cortisol-induced metabolic abnormalities includes many mechanisms, mainly impaired insulin sensitivity, altered lipid metabolism, enhanced adipogenesis and gluconeogenesis; gluconeogenesis is increased due to increased hepatic phosphoenolpyruvate carboxykinase (PEPCK) and glucose-6-phosphatase enzymes activities (Radahmadi et al., 2006; Park et al., 2011).
The altered activities of key enzymes such as glucose-6-phosphatase and fructose-1,6-bisphosphatase of carbohydrate metabolism significantly (p <0.05) increased whereas hexokinase, pyruvate kinase, glucose-6-phosphate dehydrogenase and glycogen content significantly (p <0.05) decreased in the liver of diabetic rats and also increased activities of aspartate transaminase (AST), alanine transaminase (ALT) and alkaline phosphatase (ALP).
Administration of 20-OH-ecdysone showed significant increase in the levels of glycolytic enzyme (hexokinase) and hepatic shunt enzyme (glucose-6-phophate dehydrogenase) whereas significant decrease in the levels of gluconeogenic enzymes (glucose-6-phosphatase and fructose-1,6-bisphosphatase) in diabetic treated rats.
As reviewed by Van Dam (2006), chlorogenic acid has been described as increasing hepatic glucose uptake as well as reducing hepatic glucose output by inhibiting glucose-6-phosphatase. Another hydroxycinnamic acid, ferulic acid, was also shown to increase glycogen synthesis in db/db mice (Jung et al., 2007).
We also examined the gene expression of the gluconeogenesis enzymes glucose-6-phosphatase (G6Pase) and phosphoenolpyruvate carboxykinase (PEPCK) in the liver (data not shown).
It has been suggested that increased gluconeogenesis is a main source of increased hepatic glucose production and that the ability of insulin to regulate transcription of the rate-controlling gluconeogenic enzymes, phosphoenolpyruvate carboxykinase (PEPCK) and glucose-6-phosphatase may contribute to this problem.
Synthesis of glucosamine from glucose is an insulin-dependent pathway despite going through glucose-6-phosphatase. It is therefore conceivable that, in insulin deficiency as in diabetes mellitus glucose is redirected to an insulin-dependent pathway.
Palmitate could also stimulate the transcription of gluconeogenic enzymes such as glucose-6-phosphatase (G6Pase) and PEPCK (Xu et al., 2007).
Meanwhile, methylswertianin and bellidifolin increased hepatic glycogen content, decreased glucokinase (GK) activities and increased glucose-6-phosphatase (G6Pase) activities.
Hepatic alkaline phosphatase and g-glutamyltransferase activities were significantly reduced in animals fed with SP + E diet and glucose-6-phosphatase activity was significantly enhanced.

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