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n. pl. her·pes·vi·rus·es
Any of a family of DNA viruses that form characteristic inclusion bodies within the nuclei of host cells and cause diseases such as chickenpox, infectious mononucleosis, herpes simplex, and shingles.


(Pathology) any one of a family of DNA-containing viruses that includes the agents causing herpes, the Epstein-Barr virus, and the cytomegalovirus


(ˈhɜr pizˌvaɪ rəs)

n., pl. -rus•es.
any DNA-containing virus of the family Herpesviridae, members of which cause several kinds of diseases, as chickenpox and shingles.


n (pl -ruses) herpesvirus m; human — 6 (8, etc.) herpesvirus humano 6 (8, etc.)
References in periodicals archive ?
Once a person has been infected with a herpesvirus, the virus persists in a latent form, sometimes reactivating to cause recurrent disease.
The M-Nested PCR for herpesvirus was performed based on the procedure of Cassas et al.
Serological evaluation of bovine herpesvirus 1 and 5 in cattle-breeding systems on Colombia's high plains
In the current study the Koi herpesvirus (KHV) was used as a model member of the Alloherpesviridae and rainbow trout as a model salmonid host, which were infected with KHV by immersion.
Macacine herpesvirus 1 (MaHVI; also known as B virus) is a zoonotic pathogen that is enzootic among macaque (Macaca spp.
Advances in understanding of the pathogenesis, epidemiology, and immunological control of equine herpesvirus abortion.
Serology disclosed IgG (1/160) and IgM (1/320) antibodies against human herpesvirus 7 (HHV-7) and IgG antibody against human herpesvirus 6 (HHV-6) (1/40).
In 19 chapters, medical researchers from North America, Europe, and Asia discuss the diagnosis and clinical management of human herpesvirus 6A and 6B (HHV-6A and HHV-6B) and 7 (HHV-7).
While the causative agent of AIDS was determined in 1984, it was not until a decade later that the viral etiology of KS, the Kaposi sarcoma-associated herpesvirus (KSHV; or HHV-8)) was identified.
Human herpesvirus 6 (HHV-6)5 latently infects more than 90% of adults (1) and reactivates in 30%-50% of transplant recipients (1).
This key protein is required for the virus to grow and is the target for the potent antiviral medicines used today to control many herpesvirus infections.
A plasmid was constructed that expressed fusion proteins of REV envelope (env) and VP22 of Gallid herpesvirus 2 or REV gag and VP22.