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Related to hyalinosis: malalignment, transvesical


n. hialinosis, degeneración hialina.
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Interestingly, efferent arteriolar hyalinosis was reported that specific kidney changes to DKD had a clear correlation with urine albumin excretion and disease progression.
Histological examination of the circumcision tissue usually confirms the diagnosis, showing hyperkeratosis with follicular plugging, atrophy of the stratum spongiosum Malpighi with hydropic degeneration of basal cells, lymphedema, hyalinosis and homogenization of collagen in the upper-dermis, and inflammatory infiltration in the mid-dermis.
Mitochondrial alterations are also observed in arteriolar hyalinosis [85] as well as in steroid-resistant nephrotic syndrome [86].
Lipoid proteinosis (LP), also known as Hyalinosis cutis et mucosae or Urbach-Wiethe disease, is an uncommon, autosomal, recessively inherited disorder.
Thickening of the glomerular basement membrane is the first change that can be quantified afferent and efferent glomerular arteriolar hyalinosis can also be detected with three to five years after the onset of diabetes glomerular epithelium podocyte cell structure and number are also related to glomerular permeability alterations in diabetes.
Global glomerular sclerosis and glomerular arteriolar hyalinosis in insulin dependent diabetes.
Figure 2 shows a light microscopic examination of kidneys from diabetic animals revealing diabetic glomerulopathy characterized by thickening of the glomerular basement membrane, mesangial matrix expansion, arteriolar hyalinosis, and large hyaline proteinaceous droplets (arrow) within the glomeruli (H&E, 200x), occluding capillary loops and attached outside the Bowman's capsule.
focal segmental hyalinosis and IgA nephropathy, no disease recurrence is so common as to preclude a patient from transplant.
Lipoid proteinosis (LP) was first described by Urbach and Weithe in 19291 also called Urbach-Weithe disease or hyalinosis cutis et
The perihilar variant is defined by the presence of at least 1 glomerulus with perihilar hyalinosis with or without sclerosis with >50% of affected sclerotic glomeruli possessing these perihilar lesions [13].
IF (for IgA, IgG, IgM, C3, Clq, k, and X) was performed to determine the presence of hyalinosis, capillary collapse, or immune complex deposits in the segments of glomerulosclerosis and rule out other causes of NS.