Thrombosis can be caused by the natural hypercoagulable
state during post-partum period, hypokinesis and ventricular dilation.4 Acute limb ischaemia can also be the initial presenting symptom of PPCM and should be kept in mind while managing postpartum patents.5 Moreover, the manifestation of acute limb ischaemia threatening the loss of a limb can also be the initial presentation of PPCM, as reported by Paul J.
TEG revealed a hypercoagulable
state with no platelet dysfunction (Figure 2).
Calcified right atrial wall thrombus (RAWT) is a rare entity and is usually associated with intra cardiac catheters, malignancy and hypercoagulable
Patients were excluded for the following reasons: any malignancy; hypercoagulable
state not resulting from liver disease; management with oral contraceptives, anticoagulation agents, or antiplatelet agents; uncontrolled hypertension or peptic ulcer disease; hemorrhagic stroke; or human immunodeficiency virus infection.
Although the exact cause is unclear, systemic diseases, such as hypercoagulable
states, may predispose vessels to develop occlusion.
Thus, thrombocytopenia or hypercoagulable
state is not associated with any blood group antigen.
Several mechanisms were proposed to explain why oral contraceptives increase stroke risk during the research, including raising blood pressure and making blood hypercoagulable
. 'The ideal drug is one with the lowest estrogen and progestin doses that will be effective in preventing pregnancy while minimizing adverse effects,' said neurologist Sarkis Morales-Vidal.
There were no vascular risk factors except PNH found in this patient including hypercoagulable
states, oral contraceptive use, paradoxical emboli related to an intracardiac shunt (patent foramen ovale or atrial septal defect), atherosclerotic risk factors, or cardiac arrhythmias.
Bland thrombus due to a hypercoagulable
state is more common than direct tumor extension into vessels.
laboratory studies were normal, including anticardiolipin antibody IgG and IgM, factor V Leiden, prothrombin G20210A mutation, thrombin time, antithrombin III, and protein C and S activity.
Livedoid vasculopathy is caused by a hypercoagulable
state, leading to obstruction of small dermal blood vessels by intraluminal thrombi.
It has been previously reported at various sites including the cerebral venous sinuses, inferior vena cava (IVC), hepatic veins and extremities as the deep veins of lower limbs and venous thrombosis attributed to the inflammation and hypercoagulable