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n. (used with a sing. verb)
1. The study of the interrelation between immunity to disease and genetic makeup.
2. The branch of immunology that deals with the molecular and genetic bases of the immune response.

im′mu·no·ge·net′ic adj.
im′mu·no·ge·net′i·cist (-ĭ-sĭst) n.


(Genetics) (functioning as singular) the study of the relationship between immunity and genetics
ˌimmunogeˈnetic, ˌimmunogeˈnetical adj


(ˌɪm yə noʊ dʒəˈnɛt ɪks, ɪˌmyu-)

n. (used with a sing. v.)
1. the branch of immunology dealing with the study of immunity in relation to genetic makeup.
2. the study of genetic relationships among animals by comparison of immunologic reactions.
im`mu•no•ge•net′ic, im`mu•no•ge•net′i•cal, adj.


1. the branch of immunology that studies immunity in relation to genetic formation.
2. the study of genetic relationships between animals by comparing immunological reactions. — immunogenetic, adj.
See also: Medical Specialties
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References in periodicals archive ?
[6.] Burninova LV, Moiseykina LG, Bagirov VA, Buvaeva NV (2010) Immunogenetic assessment of Kalmyk cattle.
Immunogenetic characteristics of patients with autoimmune gastritis.
Due to the intensive immunological and immunogenetic investigations of reactive arthritis over the past decades, current understanding of the mechanism of this disease is mainly based on the possible role of immune response [11, 12].
However, differences in antigen expression among parasite isolates or immunogenetic variations among hosts could determine the ability for antigen recognition by the various animal species.
Previous attempts to find common host immunogenetic factors among C.
Clinical, serologic and immunogenetic studies in patients with chronic cutaneous (discoid) lupus erythematosus who have verrucous and/or hypertrophic skin lesions.
Immunogenetic studies are urgently needed to better understand the immunologic mechanisms driving WNS-associated population declines among bat species in North America.
Immunogenetic factors are important in two ways; one by influencing the disease penetration and secondly by the production of Autoantibodies [1].
The factors determining this variability could be due to: differences in the definition of relapse, in the initial bacillary index (BI), in the follow-up time of patients, the method of gathering information and possibly immunogenetic variation in different populations.
On the basis of the results of complex study of different immunogenetic, immunologic, biochemical and neurophysiological factors and guided by clinical signs of disease we identified special, scientifically-based groups for further correcting therapy.
(12) Previous studies comparing patients with psoriatic arthritis and SAPHO describe clinical similarities; however, they fail to demonstrate a common immunogenetic background.