inotropic

(redirected from inotropism)
Also found in: Medical.
Related to inotropism: positive inotropic agent

in·o·tro·pic

 (ē′nə-trō′pĭk, -trŏp′ĭk, ī′nə-)
adj.
Affecting the contraction of muscle, especially heart muscle: an inotropic drug.

[Greek īs, īn-, tendon, sinew; see wei- in Indo-European roots + -tropic.]

inotropic

(ˌɪnəˈtrɒpɪk; ˌaɪnə-)
adj
(Pharmacology) affecting or controlling the contraction of muscles, esp those of the heart: inotropic drugs.
[C20: from Greek, from is (stem in-) tendon + -tropic]

i•no•trop•ic

(ˌi nəˈtrɒp ɪk, -ˈtroʊ pɪk, ˌaɪ nə-)

adj.
influencing the contractility of muscular tissue.
[1900–05; < Greek īno-, comb. form of is fiber, sinew + -tropic]
Translations

in·o·tro·pic

a. inotrópico-a, que afecta la intensidad o energía de las contracciones musculares.
Mentioned in ?
References in periodicals archive ?
This phenomenon suggests that mechanisms controlling the cardiovascular apparatus can successfully manage to maintain SV in the face of an impaired pre-load and thanks to the modulation of cardiac inotropism (Crisafulli et al.
2+] from intracellular stores leads to an increase of heart rate and cardiac inotropism and to vasodilatation [36-38].
Because the nervous system regulates cardiac chronotropism and inotropism as well as vascular tone, it can also regulate SBP and DBP.
In fact, it was verified that R(+)-pulegone causes negative inotropism in mammalian myocardium that was related to blockage of L-type [Ca.
7] In vitro, ghrelin decreases inotropism and lusitropism.
When the patient "moves" in phase 1 of the curve, preload is directly proportional to the systolic volume; the variations of pulse are broad, indicating that the patient is dependent on the preload or that the patient will respond to the volumen (2,3,10) (responder or dependent on volume), while if the patient is moved to phase 2 of the Starling curve the variations in pulse pressure are minimal, indicating that the patient will not respond to the liquid infusion or that what is probably required is to strengthen cardiac inotropism to increase cardiac output and avoid an overload of liquids that can lead to pulmonary edema (2,3,10) (non-responder or not dependent on volume).