kinin


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ki·nin

 (kī′nĭn)
n.
Any of various structurally related polypeptides, such as bradykinin, that act locally to induce vasodilation and contraction of smooth muscle.

[Short for bradykinin.]

kinin

(ˈkaɪnɪn)
n
1. (Biochemistry) any of a group of polypeptides in the blood that cause dilation of the blood vessels and make smooth muscles contract
2. (Botany) another name for cytokinin
[C20: from Greek kin(ēma) motion + -in]

ki•nin

(ˈkaɪ nɪn, ˈkɪn ɪn)

n.
2. any of a group of hormones, formed in body tissues, that cause dilation of blood vessels.
[1950–55; (cyto) kin (esis) + -in1]
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.kinin - any of a class of plant hormones that promote cell division and delay the senescence of leaves
growth regulator, phytohormone, plant hormone - (botany) a plant product that acts like a hormone
References in periodicals archive ?
Release of histamin, kinin and prostaglandins during carrageenin induced inflammation of the rats.
Evidence for participation of B1 and B2 kinin receptors in formalin-induced nociceptive response in the mouse.
Immediate pain probably results from a direct irritant effect whereas delayed pain probably results from an indirect effect via the kinin cascade.
vivax enfeksiyonlari kinin, doksisiklin, atovakuon/proguanil, meflokin veya artemisin bazli kombine tedaviler ile tedavi edilebilir (8).
Classification of the kinin receptor family: from molecular mechanisms to pathophysiological consequences.
Calixto, "Inflammatory muscle pain is dependent on the activation of kinin [B.sub.1] and [B.sub.2] receptors and intracellular kinase pathways," British Journal of Pharmacology, vol.
Pruneau et al., "Release of bradykinin and expression of kinin B2 receptors in the brain: role for cell death and brain edema formation after focal cerebral ischemia in mice," Journal of Cerebral Blood Flow & Metabolism, vol.
The pathophysiology of tPA-induced angioedema is not fully understood but is thought to be due to activation of both the kinin pathway as well as the complement pathway.
[13] These visceral reflexes are also evoked by chemicals such as kinin, histamine, prostaglandin, and serotonin, which stimulate the high threshold cardiopulmonary receptors to produce apnea, hypotension, and bradycardia.
For ACE gene, D allele is associated with increased ACE activity, which converts angiotensin I to angiotensin II (potent vasoconstrictor) and degrades bradykinin (vasodilator), while allele I is associated with increased endurance performance and increased kinin activity.
Denizanasina maruziyet sonucu olusan agrinin, zehirde bulunan kinin ve kinin-benzeri faktor gibi endojen veya eksojen kimyasal mediyatorlerden kaynaklanabilecegi dusunulmektedir.