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 (lĭp′ō-pŏl′ē-săk′ə-rīd′, lī′pō-)
Any of a group of polysaccharides in which a lipid constitutes a portion of the molecule, found in the outer cell wall of gram-negative bacteria.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.


(ˈlaɪp oʊˌpɒl iˈsæk əˌraɪd, -rɪd, ˈlaɪ poʊ-)

any of a class of polysaccharides to which lipids are attached.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
References in periodicals archive ?
Early pulmonary lesions in turkeys produced by nonviable Aspergillus fumigatus and/or Pasteurella multocida lipopolysaccharide. Avian Diseases 42 770780.
Outer membrane protein and lipopolysaccharide heterogeneity among Eikenella corrodens isolates.
Components of whole-cell vaccines, including lipopolysaccharides and internal proteins, were important in signaling the innate immune system to shape a more robust response.
Although, MAb subtyping may not be reliable since the expression of the lipopolysaccharide types that react with the MAb is prone to phase variation and may also depend on the growth stage [26], it was shown recently that the MAb 3/1 epitope is expressed during all growth phases [27].
Lipopolysaccharide (Escherichia coli LPS, serotype 0127:B8) and Vitamin E were purchased from Sigma Chemical Co.
"Our data highlight that these people can be exposed to as much as 126% more circulating lipopolysaccharide after a high-fat meal," said Dr.
Lipopolysaccharide exposure induces inflammation in bovine cumulus-oocyte complexes
peregrina seed extract, which might be used as a natural drug for treatment of inflammatory-related disease, on NO and pro-inflammatory cytokine IL-1[beta] production in lipopolysaccharide (LPS)-induced macrophage cell line J774A.1.
Protective effect of resveratrol against acute lung injury induced by lipopolysaccharide via inhibiting the myd88-dependent Toll-like receptor 4 signaling pathway.
In the study, the researchers injected mice with lipopolysaccharide (LPS) - a non-infectious bacterial toxin that causes symptoms associated with sickness.
* Lowered the resulting elevated circulating lipopolysaccharide levels, and
TLR4 is a member of the toll-like receptor family and can recognize lipopolysaccharide (LPS), the main component of gram-negative bacteria.