melanoblast


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mel·a·no·blast

 (mĕl′ə-nō-blăst′, mə-lăn′ə-)
n.
A precursor cell of a melanocyte or melanophore.

mel′a·no·blas′tic adj.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

melanoblast

(ˈmɛlənəʊˌblæst)
n
(Physiology) a cell that develops into a melanocyte
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.melanoblast - an epidermal cell that is a precursor of a melanocyte
epidermal cell - any of the cells making up the epidermis
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.
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(2008) The secreted metalloprotease ADAMTS20 is required for melanoblast survival.
Melanomas are benign tumors arising from pigment producing melanocytes, which are matured forms of melanoblast that arises from neuro-ectoderm during development.
They can be hereditary or acquired and have many causes, including abnormalities in melanoblast migration, melanosome development or transfer, changes in the numbers of melanocytes, or defects in melanin synthesis [1, 2].
determined that P-Rex1 deficiency suppresses melanoblast migration and development of metastases [10].
Excessive amounts of HQ can kill the melanoblast and cause skin disease called Vitiligo [5, 6].
Although its fully validated function is unknown but it is believed that it may be required for melanocyte differentiation on the basis of its expression pattern in melanoblast and melanocyte in human fetal cells.
Two members of the Gaq class of G-protein [alpha] subunits, Gnaq and Gna11, proteins involved in signaling by G-protein-coupled receptors, emerged as the most important molecules controlling early melanoblast proliferation in the dermis.
Stimulation of melanoblast pigmentation by 8-methoxypsoralen: the involvement of microphthalmia-associated transcription factor, the protein kinase a signal pathway, and proteasome-mediated degradation.