SIBO causes malabsorption across the intestinal microvillus
membrane as a result of damage to enterocytes, as well as impaired digestion in the intestinal lumen.
Cryptosporidium is a coccidian parasite which resides in the microvillus
of the lumen and multiply in the microvillus
producing Oocyst, leading to malfunction of the lumen.
This is in accordance with previous research work in which hyperglycaemia suppresses the cell cycle leading to slowing of the growth in chick embryos.10 Alteration of metabolites could be the basis of this growth inhibition as shown by work done by Moley and fellows by exposure of pre-implantation mouse embryos to elevated glucose.9 Excess glucose in mammalian post-implantation embryos resulted in ultra-structural changes in visceral yolk sac, such as reduced microvillus
surface, fewer mitochondria and a sparser distribution of endoplasmic reticulum.
The tegument covering the anterior adhesive areas is microvillus
The enterocytes were columnar, with an electron dense cytoplasm, a basal located granular nucleus, and microvillus
towards the lumen (Fig.
is an apicomplex, intracellular, extracytoplasmic protozoan parasites which infect the microvillus
epithelium of gastrointestinal tract in animals, birds, reptiles and man.
The cross section area of each microvillus
is given by
Histopathological findings in study cases from these farms confirmed the bursitis identified at necropsy, showing hyperplasia of bursal epithelial cells, with various life-cycle stages of Cryptosporidia parasitizing the microvillus
In low dose group, normal structure of fetal type II pneumonocyte was observed and microvillus
outstanding cell surface fell off a little with clear cellular nuclear structure.
was preserved at apical portions of the corneal epithelium of MSCs-treated rats and there was no prominent sign of cellular injury at cornea of these rats (Figures 4 and 5).
jii visualizes two types of receptor cells (i) Ciliated sensory with microvillus
(ii) Ciliated non-sensory cells.
MYO5B mutations cause microvillus
inclusion disease and disrupt epithelial cell polarity.