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n. pl. my·co·bac·te·ri·a (-tîr′ē-ə)
Any of various rod-shaped, aerobic, often pathogenic bacteria of the genus Mycobacterium, including the causative agents of tuberculosis and leprosy.

my′co·bac·ter′i·al adj.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.


(Medicine) of, relating to, or caused by mycobacteria
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014
References in periodicals archive ?
Timely surgical intervention for mycobacterial empyema is both safe and effective for the diagnosis and treatment of this disease.
These opportunistic pathogens account for an increasing proportion of mycobacterial infections [2-4].
In recent years, the BACTEC MGIT 960 system, a fully automated and nonradiometric culture system, has been recommended for faster mycobacterial isolation from clinical specimens [4].
Other reasons to biopsy include suspected infection --whether bacterial, fungal, or mycobacterial; atypical appearance; suspected vasculitis; and recent travel history.
Susceptibilities to mycobacterial osteoarticular infections are thought to result from disorders in Type 1 T-helper-cell-mediated immune inflammation, resulting in deficient production and/or action of interleukin 12 (IL12) and interferon gamma (IFN-[gamma]) [1].
Mycobacterial, fungal and other opportunistic infections force the clinician to rule out a large list of conditions associated with secondary immunodeficiency as infectious agents (HIV, Herpesvirus, HTLV), drugs (steroids, immunosuppressants, biologics, chemotherapy), metabolic diseases (diabetes, renal failure, cirrhosis), malignancies (leukemia, lymphomas and solid tumors) and environmental conditions (radiation, heavy metals) (2) but even adult patients can have late onset primary genetic immunodeficiency disorders as chronic granulomatous disease, X-linked agammaglobulinemia, interleukin-12 receptor deficiency or interferongamma (IFN-?) and interleukin-23/interleukin-17 pathway defects (3) explaining their pattern of infection or the presence of opportunistic microorganism.
These methodologies allow for the rapid identification of mycobacterial organisms in less than one day (12).
Parotid gland infections are usually caused by pathogens such as Staphylococcus aureus and anaerobic bacteria [1] and only in rare cases are secondary to a mycobacterial infection.
Among A Group of patients (30 in nos.), 27 had atypical mycobacterial infections and 3 patients had nonmycobacterial infections.
(5,6) An inherent impaired immunity against both mycobacterial organisms has been postulated as the aetiology for dual infection; however, it appears that anergy in leprosy is pathogen specific.
Mycobacterial infections, such as Mycobacterium tuberculosis or nontuberculous (atypical) mycobacterial (NTM) species, often present as indolent infections refractory to treatment with antibiotics.