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Related to nicotinic: nicotinic acid


 (nĭk′ə-tĭn′ĭk, -tē′nĭk)
1. Of or relating to nicotine.
2. Of or relating to niacin.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.


(ˌnɪk əˈtɪn ɪk, -ˈti nɪk)

1. of, pertaining to, or containing nicotine.
2. related to or imitating the effects of nicotine.
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.


[ˌnikəˈtɪnɪk] ADJ nicotinic acidácido m nicotínico
Collins Spanish Dictionary - Complete and Unabridged 8th Edition 2005 © William Collins Sons & Co. Ltd. 1971, 1988 © HarperCollins Publishers 1992, 1993, 1996, 1997, 2000, 2003, 2005
References in periodicals archive ?
LAHORE -- Qaneta Irfan, a student of the Chemistry Department of the Lahore College for Women University (LCWU), has made a herbal tea using Moringa Oleifera leaves.The leaves of Moringa Oleifera are rich in minerals like beta-carotene of Vitamin A; Vitamin B such as folic acid, pyridoxine and nicotinic acid; vitamin C; D and E.
Presently, the product is in phase two trial to treat agitation and is a negative allosteric modulator of the alpha-7 (alpha-7) nicotinic acetylcholine receptor.
Nicotinic acetylcholine receptors, or nAChR, associated with acetylcholine-mediated neurotransmission have been linked to several neurological conditions and public health issues, notably tobacco addiction.
The study reveals that exercise during nicotine exposure markedly reduces the severity of nicotine withdrawal symptoms, an effect that is accompanied by increased activation of a7 nicotinic acetylcholine receptors (which are targets of nicotine) in the hippocampal region of the brain.
Vitamin [B.sub.3] occurs naturally in two forms: niacin (also called nicotinic acid) and niacinamide (also called nicotinamide).
Some authors have reported that nAChRs stimulation of the brain dopamine receptor system normalizes a number of sensory processing deficits associated with schizophrenia, including sensory, PPI and eye- tracking deficits, the hypothesis of nAChR "hypofunction" in schizophrenia has stimulated the development of selective nicotinic receptor agonists as putative treatments for negative symptoms and cognitive dysfunction.
We provide evidence that the effect of CCL3 is mediated via CCR1 and calcium-independent phospholipase A2 (iPLA2), release of a soluble small mediator and nicotinic acetylcholine receptors (nAChR) containing subunits a7 and a9.
It is characterized by decrease in the number of available nicotinic acetylcholine receptors at postsynaptic membrane due to presence of pathogenic autoantibodies [1, 2].
The protein, called alpha-4-beta-2 nicotinic receptor, sits on nerve cells in the brain.
Patients exhibit muscarinic and nicotinic symptoms depending upon severity of compounds.